Guest viewing limit reached
  • You have reached the maximum number of guest views allowed
  • Please register below to remove this limitation
  • Already a member? Click here to login
  • 💪 Hello, please SIGN-UP FOR A FREE account and become a member of our community!
    You will then be able to start threads, post comments and send messages to other members. Thanks!
  • 🚨Muscle Gelz HEAL - A Topical Peptide Repair Formula with BPC-157 & TB-500!🚨

Why SOME lose hair via Dihydrotestosterone

RawCutlery

Be Raw, Be Cut, Raw Cutlery
Registered
Joined
Jul 31, 2025
Messages
89
Reaction score
249
Points
41
Age
39
Location
Central Florida
Good morning everyone, let’s jump right into this..

Hair loss from androgenics, anabolics, testosterone derivatives, or DHT-based compounds comes down to a tug-of-war between genetics and hormone chemistry.

The hormone culprit: DHT
Testosterone gets converted by the enzyme 5-alpha reductase into dihydrotestosterone (DHT). DHT is way more potent than testosterone in terms of binding to androgen receptors. When DHT locks onto receptors in hair follicles especially on the scalp’s crown and temples, it tells the follicle to shrink. From there a process called miniaturization takes place. Over time, the hairs grow back thinner and shorter, until they stop showing up at all. 😩😩

FF528E0D-83A6-4997-B4E0-5B8B17FBD77F.jpeg

Genetics load the gun, androgens pull the trigger
Not everyone loses hair, even on heavy androgenics. This is because it depends on how sensitive your follicles are to DHT. If you inherited a strong predisposition to male pattern baldness, you’ll notice shedding much faster once you raise androgen levels. If you didn’t, you might run cycles without much effect on your hairline.

Follicle battlefield: scalp vs. body
DHT is a paradox, on the scalp, it shrinks follicles, but on the body ace, chest, arms it stimulates them. That’s why guys on androgenics might lose their head hair while suddenly sprouting a thicker beard or chest forest. It’s all about the distribution of androgen receptors.


Acceleration, not invention
Androgenics don’t create baldness they accelerate the timeline if you’re predisposed. A man who would naturally thin out in his 40s may see it happen in his mid to late 20s if he’s running high-androgen cycles.

The “protection” strategies
Some try to fight this by blocking DHT conversion with drugs like finasteride Propecia or dutasteride. Also by simply using topical blockers like minoxidil. These can slow the loss, but they come with their own risks, especially when mixed with supraphysiological androgen levels.

IMG_6073.jpeg
In short, androgenics cause hair loss because DHT binds to sensitive follicles on the scalp, shrinks them, and speeds up a genetically programmed process. It’s basically your DNA’s hair calendar being set to “fast-forward.”

I know a lot of you are aware, but there are many that don’t understand why and how it happens.

I’m gonna be getting my 3rd masters degree in Kinesiology, I start the 25th of this month. I plan to then put the three together, my MBA, masters of Business Intelligence and Masters of Kinesiology to have a very strong package to present to potential employers.
 
Last edited:
Just curious to what the risks are.

Is what your referring to loss of libido? And impacts on mood?

It would seam that testosterone does everything that DHT would do. Especially when your have super physiological levels of free T. DHT Seams only critical for puberty. If anything low DHT is better for your prostate.

From the research I’ve done it doesn’t offer any protection or benefit to any of the major organs.

Testosterone in is pure form is more for that.

Wouldn’t it be better to reduce DHT while on cycle.

Plus the side effects only occur within like 2% of men.
 
Yeah the libido and psychological effects..

Also reducing it can slightly decrease muscle hardness, strength, or recovery capacity.

Most men maintaining normal testosterone levels can offset this through training and nutrition.

It’s an interesting biological balancing act.

Preserving hair versus preserving hormonal vigor. In essence, you’re shifting the distribution of androgens, not eliminating them, so the art is in controlling the shift without dulling the system’s edge.

Thanks for contributing to this thread.
 
Last edited:
Yeah, no problem. Shouldn’t test in super physiological dosages counteract the loss of the benefits derived from DHT? Such as mood and libido?
 
And wouldn’t eliminating DHT allow more testosterone to make it through to the nucleus, giving you even more androgenic effects from testosterone?
 
Yeah, no problem. Shouldn’t test in super physiological dosages counteract the loss of the benefits derived from DHT? Such as mood and libido?

Even if total and free testosterone skyrocket, if 5-alpha reductase is blocked, much less of it converts into DHT.

The androgen receptor activation profile changes. You get more anabolic signaling (muscle, recovery) but less of that sharp, driven “DHT effect” on libido and mood.

Some men feel fine; while others describe feeling emotionally flat, and not there sexually.

Supraphysiological testosterone can mitigate some loss of libido or mood caused by low DHT, but it won’t fully replicate DHT’s qualitative effects, especially on mental sharpness, sexual sensation, and assertiveness.

The relationship between testosterone, DHT, and mood is nonlinear, therefore more isn’t always better; balance is the key
 
the studies show that testosterone replicates most if not all of what DHT does. I believe the only difference is that DHT binds much stronger to the androgen receptors therefore being 6-8 times more effective than testosterone. Which would explain why if blocked off it can cause negative symptoms.

However side effects are only found in 2% of men. So I wouldn’t say that’s a good argument to prove that the loss of libido and flattening of the mood is because of DHT. These people are the outliers. It wouldn’t say it’s the rule.

The fact that’s 98% don’t feel these symptoms especially men with super physiological doses of T, would indicate that Free T replicates exactly what DHT would.

I watched a More plates more dates video where he sites a study where the people who had no dht and who had dht had no significance difference in muscle growth.

So I would say that taking a 5a predicate inhibitor would probobly be better if your not the exception 2%. In order to avoid prostate enlargement, acne and accelerated balding.
 
Plus when taking something like dutasteride you can have more peace of mind taking DHT derivitives. Although, dutasteride doesn’t block the “dht” from compounds like Anavar or Primo, because they are already in “dht” from when they hit the bloodstream they bypass the enzyme, you will still have far less “dht”. Plus, Anavar and primo arnt as powerful as DHT. They are much less potent. So the effect will be minimal on prostate, acne, hair loss.
 
Plus when taking something like dutasteride you can have more peace of mind taking DHT derivitives. Although, dutasteride doesn’t block the “dht” from compounds like Anavar or Primo, because they are already in “dht” from when they hit the bloodstream they bypass the enzyme, you will still have far less “dht”. Plus, Anavar and primo arnt as powerful as DHT. They are much less potent. So the effect will be minimal on prostate, acne, hair loss.
I respect your knowledge, chime in/contribute more often. I love these types of conversations. We went back and forth without being disrespectful, I love this bro!!
 

Latest threads

Back
Top