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What is your go-to AI & why? Tamoxifen, Raloxifene, Proviron & Mast included...

No more than 25mgs Aromasin pw for me all the way up to about 2000 migs per week (1600 migs Aromatizable)

If someone was using something as simple as test, primo, anavar, then I'd say no AI at all if the test isn't super high and even then, you could up the Primo and possibly still be OK.

It all depends on the compounds used, the dosages used, and how each individual reacts to those doses.

I may need 12.5 Aromasin pw and another person may use 50mgs per week on the same dosage.

Sent from my moto g 5G (2022) using Tapatalk
 
Yeah I'm big into nutrition and wanted to do some write ups but just haven't got around to it. Need to structure my time better.

For a good way to take a 1:1 ratio of test to dht. 200mg of test could use 200mg primo or masteron. I also like to run 25-50mg of provi a day regardless. I also want to start using 5-10mg of aromasin ED. Perhaps if using something that aromatizes like tren or dbol you could incease aromasin dosages and add nolvadex to stop gyno. Otherwise I might just start using nolvadex once my puffy nips are gone just once or twice a week.
Hey brother there's a ton of write-ups that I got, the I've actually taken down from the stickies that were all over the place throughout the years, some of these prior to you joining. Most of these prior to when you were joining, there is a time I would just write a lot of literature, and I would centrally focus on studies, and authors, who supported the studies where did the finances come from, I would peel back the layers.. It may appear that this is something new but it's not I assure you... I'm known for always writing something, whether it's factual or a matter of opinion, but I like to give my take and my interpretation with the breakdown of what they're saying.

There's a bunch more stuff that I'm going to start refreshing that was before you join date, then I'm going to start updating a lot of those because my mindset has changed with a lot of protocols.
 
Yeah I'm big into nutrition and wanted to do some write ups but just haven't got around to it. Need to structure my time better.

For a good way to take a 1:1 ratio of test to dht. 200mg of test could use 200mg primo or masteron. I also like to run 25-50mg of provi a day regardless. I also want to start using 5-10mg of aromasin ED. Perhaps if using something that aromatizes like tren or dbol you could incease aromasin dosages and add nolvadex to stop gyno. Otherwise I might just start using nolvadex once my puffy nips are gone just once or twice a week.
Brother writing is like a full time job, one of the reasons for my "many" typos, just don't have the time to go back and grammar check things that auto spell check destroys on me.
It's fun, but yet writing can suck because so many articles look the same, yet we have to use different wording.. Like talking about gear and sides, everyone means well, but the internet is full if replies that LOOK the same.. How to be unique? know what I mean.. :unsure: :cautious:

That's like writing about basic bird-house building (great analogy right hahah).. How to make it NOT look like 50 years worth of books, reads ,info and DYI's ?!?!?!
Let me know if I can help anywhere with writing brutha, would love to assist.. Writing is my passion, truly! :giggle::geek::daydream:
 
trenbolone does not aromatize
Tren does not cause gyno in the sense that we are using it in the argument sense of estro gyno, but its simply the go to word that people use to get a point across..

Tren can in fact in some sensitive people inflame the PR's, tren is a progesterone which is an agonist to the PR's...
We all get hung up on estro stimulation and development within the mammary duct tissue, yet progesterone stimulations the proliferation of what we all need to remember "secretory tissues".. The pituitary has a huge role here..
There is over 7 endocrine hormones/secretions that can induce this and believe it of NOT HGH is one of them...
At this molecular level, biology has shown by way of biosynthesis the variety and multi-steps what we could call "gym rat back door mechanism of action with unrelated estro gyno"
There's a slew of enzyme-catalyzed processes that could take place where MANY-MANY-MANY substrates are converted into more complex interplay, where such aforementioned hypothesis - anecdotal accounts mention, Tren, nandrolone, drol and at times HGH progesterone receptor upregulation synthesis with tenderness and possible prolactin around mammo/glandular tissue
RECs are almost always being upregulated.. PRs under-go upregulation synthesis by pituitary signaling ((( Tren has a HIGH affinity for PRs)) thus there can be an activation, at times activating yet downregulating glandular tissue (this is where people will have swelling but no discharge and if there was it would be dark in color almost blood looking, reddish/brown)

We can go all day with these "back door properties"..
 
I've been using Tamoxifen twice weekly since I started anadrol. Just as a preventative measure. It doesn't make me feel like shit or anything for that matter. 50mg a day pre workout, training days only. 500mg test cyp pinning twice weekly. January I'll be adding Deca or NPP to finish out the blast.
 
Tren does not cause gyno in the sense that we are using it in the argument sense of estro gyno, but its simply the go to word that people use to get a point across..

Tren can in fact in some sensitive people inflame the PR's, tren is a progesterone which is an agonist to the PR's...
We all get hung up on estro stimulation and development within the mammary duct tissue, yet progesterone stimulations the proliferation of what we all need to remember "secretory tissues".. The pituitary has a huge role here..
There is over 7 endocrine hormones/secretions that can induce this and believe it of NOT HGH is one of them...
At this molecular level, biology has shown by way of biosynthesis the variety and multi-steps what we could call "gym rat back door mechanism of action with unrelated estro gyno"
There's a slew of enzyme-catalyzed processes that could take place where MANY-MANY-MANY substrates are converted into more complex interplay, where such aforementioned hypothesis - anecdotal accounts mention, Tren, nandrolone, drol and at times HGH progesterone receptor upregulation synthesis with tenderness and possible prolactin around mammo/glandular tissue
RECs are almost always being upregulated.. PRs under-go upregulation synthesis by pituitary signaling ((( Tren has a HIGH affinity for PRs)) thus there can be an activation, at times activating yet downregulating glandular tissue (this is where people will have swelling but no discharge and if there was it would be dark in color almost blood looking, reddish/brown)

We can go all day with these "back door properties"..
Nailed it I think. Whole time I was worried about estrogen. I was doing high dose tren/hgh. Talked to some members in pm. I think it's prolactin related.
 
I've been using Tamoxifen twice weekly since I started anadrol. Just as a preventative measure. It doesn't make me feel like shit or anything for that matter. 50mg a day pre workout, training days only. 500mg test cyp pinning twice weekly. January I'll be adding Deca or NPP to finish out the blast.
Add NPP to finish. It's the better option with the shorter half life.
 
SB Labs
Nailed it I think. Whole time I was worried about estrogen. I was doing high dose tren/hgh. Talked to some members in pm. I think it's prolactin related.
With the combination of tren and HGH people would be baffled on hell that could be more directly related with those two than anything else, meanwhile they're throwing everything in the kitchen sink at it trying to combat it.

This is where Tamoxifen or ROLOXIFEN Will absolutely shine.
"Super smart" brand also makes a really good prolactin supplement that you can get..
Regardless of estrogen levels, even if they are tanked, when using tamoxifen or ROLOXIFEN They have a tendency with indirectly subsiding the PRs, PR's can sometimes be activated by the sensitivity through the ERs and it doesn't necessarily have to be estrogen. The ERs will sometimes act like bespoke person or the ambassador that will translate a different language, and from there it will awaken the PRs.
When the ERs are suppressed and occupied the PRs sometimes will subside as well with the swelling and inflammation.. regardless of what your estrogen is at..

Something else to keep in mind, We all know how some quick turnaround labs can have cross-reactivity with Trenbolone in your blood work coming back as elevated estrogen (clearly this is why we use a sensitive essay).. something to keep in mind that sometimes with some people their bodies can do the same thing, it will see a specific molecule, a structure of a hormone and if it closely resembles something, it will treat it as if it is that, even though it's not estrogen, the body will behave like it is, yet blood work will show otherwise...
Think of someone having a false sense of hypoglycemia, when they test their blood sugar levels they're fine but yet their body is going through all of the tell-tale signs and symptoms of having low blood sugar, it's a false indication.

Hormones can do the same thing, TRENBOLONE is super slutty and DROL can do this too..
Last but not least, HGH and some peptides are notorious for this yet people look in a different direction not knowing how to make the correlation.. The pituitary can really screw things up on people..
 
I've been using Tamoxifen twice weekly since I started anadrol. Just as a preventative measure. It doesn't make me feel like shit or anything for that matter. 50mg a day pre workout, training days only. 500mg test cyp pinning twice weekly. January I'll be adding Deca or NPP to finish out the blast.
I had a real smooth stack going with Test, NPP, Primo, no AI required and decided to spice it up a bit with 50 MG Anadrol pre workout and noticed that after about 12 hours maybe the drol starts wearing off but nips got a bit spicy so i popped a tamoxifen and next day i was fine. Wondering if i should just raise the primo or add some mast while experimenting with anadrol so i don't have to keep taking the tamoxifen. things to ponder lol.
 
Trenbolone alone does not aromatize, therefore it does not increase estrogen.
It does not increase progesterone and does not increase prolactin, although it depresses thyroid hormone levels.

A whole different story if taken in combination with aromatizable steroids.
Here the interactions increase and without adequate control of estrogen with the use of an anti-aromatase, Estradiol levels increase and with them, often but not always, there is an even high increase in Prolactin.
A combined increase in estrogen (particularly estradiol) and prolactin is very often the cause of the onset of gynecomastia.
Tren 100% raises Prolactin. You are correct that there is 0 effect on Estrogen.

I'm not sure if that was a typo Phill.

That's why people use Caber when on 19-nors.

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What is your go-to AI & why?
If you do use or had to use an AI what would it be, and why? Just for conversation...
Your personal pros and cons...
Some AI's are just terrible for your health and markers , yet the reason I'm bringing this up, some guys swear by these exact AI's that have terrible sides and others won't go near them and will look elsewhere..

We can add Tamoxifen and Raloxifene into the chat, not an AI - clearly, used to assist...

If you use mast/proviron with keeping estro low, low SHBG, higher free T and no need for AI's and so on, feel free to mention if you prefer those for whatever your reason..
12.5mg Aromasin EOD

OR

12.5mg Aromasin E3.5D with 30mg Mast Prop PD.

I think Aromasin is a superior AI when compared to others. Not only is it safer, but I think it also comes with less side effects. (One of the main ones being, it won't crush your E2 and have you feeling like a bag of dog shit)

This works with pretty much anything I run. However, I don't run more than 400mgs of any one compound and don't use more than 1200mgs-1400mgs of gear.

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