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Test E vs Test cyp

I’ve always went with test e I don’t know why just what I was introduced to
 
They're basically the same and interchangeable. Cyp has a little longer half life. 3 more days or so

UNCLE Z REP

WWW.UNCLEZ.RU FOR LIST/ORDERING
 
Cypionate has one additional carbon atom. Makes it last a bit longer than E. US uses Cyp more and Enanthate is used more worldwide.

Max
I like undecanoate now. Still want to try the Xyosted sub q pen version; just to see for 8 weeks but it is a hassle w/insurance. Imagine that! Lol

Max
 
You won’t notice a difference either Way
 
I personally like the idea of cyp being longer I have a bunch of cyp300 I love
 
No difference, but you personally might like one over the other. Try them both!


Sent from my iPhone using Tapatalk
 
i've always just used "e", but started a cycle w/ Sustanon (which has 4 diff. esters) and I think I like it better than "e". BUT- Test is test for the most part unless u use susp. or prop(no or very short esters). OR decanoate which is the longest ester-I think. Strictly a matter of preference imo. Cyp. and "e" def. the most popular. Try both, but u prob. won't notice a diff. anyway.
 
Either works just fine. 7 carbon atoms in Enanthate vs 8 in Cypionate. Package insert on RX Cyp states half life is 8 days, Enanthate is 6 or 7. Still SHOULD inject either twice weekly. This smooths out the trough/peak band.

Test undecanoate is the longest Ester, Bayer's insert states to take 1 gram (4cc) in one inject every 3 months. That's nonsense! Your peak will be 3,000 ng/dl by day 12 and you'll trough 400 ng/dl by the time you inject again. What a psycho rollercoaster! Inject undecanoate 1 cc monthly for TRT. I don't think its wise to use undecanoate for blasts as, if you start having sides your stuck with them for 1 - 2 months.
 
Test E makes you huuuuuuge, Test C turns you into a pussy.
 
.Used both and noticed no difference between the two. Cyp has a slightly longer elimination half life, but it shouldn't matter between the two esters..especially if you're pinning eod or e3d.


 
Last edited:
Can you easily switch between Test cyp and Test E? For some reason I always thought if you wanted to switch from either/or that you'll need to wait 2-4 weeks before the other ester takes effect. Never really asked anyone but assumed it by based on how people usually start in the first place when they first take test.

I've switched in the past but always took a shot of both for a few weeks before stopping one.
 
hot rod said:
i've always just used "e", but started a cycle w/ Sustanon (which has 4 diff. esters) and I think I like it better than "e". BUT- Test is test for the most part unless u use susp. or prop(no or very short esters). OR decanoate which is the longest ester-I think. Strictly a matter of preference imo. Cyp. and "e" def. the most popular. Try both, but u prob. won't notice a diff. anyway.
Click to expand...

Test is test period. The ester only affects the release time of the hormone. Test p is test , so is test c. No difference.
 
Humachine said:
Wondering what’s better or what’s the difference
Click to expand...

There can be some slight differences with getting all winded here.

Some people have what seems like an allergic response to TestE compared to TestC.
People have blamed the labs or other things as potential factors when in actuality it was most likely acidic residues left behind in the TestE. TestC at 200mg per mL (most standard dose) is a lot more user friendly, with less site irritation.
With TestE at the most popular compounding dose "250" actually is quite a high for for some with a singular injection, this is why people report better site response with drugs at 200mg/mL (depending on the ester)
I personally love test cyp simply for its smooth injection response. TestE in almost all cases gets a bit tender or lumped up for my.
There is also some differences in both E and C when you peel the layers back with half-life, Cmax levels and so on, but at the end its really small and comes down more to site comfortability.

Truth is, people will respond differently with the same ester, it all comes down to having that positive "smooth" response". If the ester is problematic at the site, this will effect the metabolization rate.

______

How one particular ester yielding different results in users.. Or, others claiming one ester to be Superior to the next.. This is all individual based on ones static system of checks/loops, no two individuals are the same.. This is why people argue that cypionate is much more potent than enanthate, as this is a fragile argument at best and makes little sense, as your muscles/receptors are only recognizing free bio hormone no matter what the ester that was utilized to deploy the parenting hormone!

These are great questions & a worthwhile topic, as there's some real truth behind this;

Let's begin with the expression of a single hormones yielding an opposite effect - rather than a positive "smooth" response to the hormone as some individuals claim/experience.. An appropriate example to begin with would be that of an alcoholic...

Why? Because of Genetics, I'll explain!

Everyone posses different gene expression, with enzymes, protein bonds and so on..(Enzymes are known to catalyze more then 5k biochemical types)..Yet our bodies posses inhibitors and activator molecules that can greatly effect the activity, by increasing or decreasing hormonal actives.. Now, that of an alcoholic, it's a genetic predisposition (same applies for particular endocrines with sensitive users concerning AAS) in which one posses a group of particular enzymes that metabolize alcohol much differently compared to other individuals, thus the effects will be much greater, and more detrimental in some individuals over the next..(Different metabolization)

Now, let's compare this to the hormones!

(Example) Testosterone can have 3 roles
1) Being testosterone as it's primary function..
2) Converting into DHT (dihydrotestosterone: a more potent metabolite/androgen)..
3)Converting into estrogen

Here you see this effect differs greatly by individual instances, as genetics, age or even ethnicity (in some cases), or even sex may be a factor, whether one has a greater presence of aromatase enzymes, or even a deficiency.. Hormones/esters have various biosynthetic pathways occurring in the endorcrine, some either or before reaching their target tissue(to control plasma levels or active compounds),or at times after termination of their actions (inactivation and elimination)..However many of hormones and esters are metabolized within their target tissue, in which a complex interplay between activation and inactivation mechanisms serve to regulate the specificity and the amplitude of the hormonal response..

This is why "singular ester" hormone treatment is NOT designed as a one size fits all therapy...

Just an FYI: There's two main types of enzymes that act in the cleaving process of the ester in which activate the hormone...These 2 particular "Enzymes" are esterases & hydrolysate.. These come along and cleave pieces of the ester off the hormone, thereby releasing the active chemical (parenting steroid hormone) and allowing it to do its job (ultimately muscle building/tissue via protein synthesis, positive nitrogen balance and so on )... Now in the bloodstream which testosterone and blood born nutrients and substances circulate is recognized as the "medium". These enzymes circulate in the medium and directly affect the release of "active" hormone in the bloodstream by ester cleavage, thus exerting different expression per individuals, trigger a chute of cellular interaction at receptor sites that will differ from one to the next..

People need to remember that hormones are "chemical messengers" that rely messages to cells that display specific receptors for each hormone and respond to the signaling.. Depending on the ester and the individuals metabolization ratio the hormone can/may make changes directly to a cell, by changing the genes that are activated, or by making changes indirectly to a cell by stimulating other signaling pathways inside a specific cell group that is effected and effect other processes, thus this can "initiate" an intracellular cascade of events..

This is why the "same/singular hormone ester" effects people differently, because there's numerous ways a hormone can yield different effects.. So, depending on which transcription factors are present different genes will be transcribed in response...Thereby the reason some individuals tend to bloat and others do NOT, as some can run 250mgs of Test and begin to see signs of fluid retention or edema exert in the circular system, or particular regions of the body.. Yet, there's individuals that can utilize 1000mgs of Test and remain dry.. Of course diet permitting.. (but this article's about biosynthesis, and not diet)

Regards,
Vision


 
Get Shredded!
Vision said:


There can be some slight differences with getting all winded here.

Some people have what seems like an allergic response to TestE compared to TestC.
People have blamed the labs or other things as potential factors when in actuality it was most likely acidic residues left behind in the TestE. TestC at 200mg per mL (most standard dose) is a lot more user friendly, with less site irritation.
With TestE at the most popular compounding dose "250" actually is quite a high for for some with a singular injection, this is why people report better site response with drugs at 200mg/mL (depending on the ester)
I personally love test cyp simply for its smooth injection response. TestE in almost all cases gets a bit tender or lumped up for my.
There is also some differences in both E and C when you peel the layers back with half-life, Cmax levels and so on, but at the end its really small and comes down more to site comfortability.

Truth is, people will respond differently with the same ester, it all comes down to having that positive "smooth" response". If the ester is problematic at the site, this will effect the metabolization rate.

______

How one particular ester yielding different results in users.. Or, others claiming one ester to be Superior to the next.. This is all individual based on ones static system of checks/loops, no two individuals are the same.. This is why people argue that cypionate is much more potent than enanthate, as this is a fragile argument at best and makes little sense, as your muscles/receptors are only recognizing free bio hormone no matter what the ester that was utilized to deploy the parenting hormone!

These are great questions & a worthwhile topic, as there's some real truth behind this;

Let's begin with the expression of a single hormones yielding an opposite effect - rather than a positive "smooth" response to the hormone as some individuals claim/experience.. An appropriate example to begin with would be that of an alcoholic...

Why? Because of Genetics, I'll explain!

Everyone posses different gene expression, with enzymes, protein bonds and so on..(Enzymes are known to catalyze more then 5k biochemical types)..Yet our bodies posses inhibitors and activator molecules that can greatly effect the activity, by increasing or decreasing hormonal actives.. Now, that of an alcoholic, it's a genetic predisposition (same applies for particular endocrines with sensitive users concerning AAS) in which one posses a group of particular enzymes that metabolize alcohol much differently compared to other individuals, thus the effects will be much greater, and more detrimental in some individuals over the next..(Different metabolization)

Now, let's compare this to the hormones!

(Example) Testosterone can have 3 roles
1) Being testosterone as it's primary function..
2) Converting into DHT (dihydrotestosterone: a more potent metabolite/androgen)..
3)Converting into estrogen

Here you see this effect differs greatly by individual instances, as genetics, age or even ethnicity (in some cases), or even sex may be a factor, whether one has a greater presence of aromatase enzymes, or even a deficiency.. Hormones/esters have various biosynthetic pathways occurring in the endorcrine, some either or before reaching their target tissue(to control plasma levels or active compounds),or at times after termination of their actions (inactivation and elimination)..However many of hormones and esters are metabolized within their target tissue, in which a complex interplay between activation and inactivation mechanisms serve to regulate the specificity and the amplitude of the hormonal response..

This is why "singular ester" hormone treatment is NOT designed as a one size fits all therapy...

Just an FYI: There's two main types of enzymes that act in the cleaving process of the ester in which activate the hormone...These 2 particular "Enzymes" are esterases & hydrolysate.. These come along and cleave pieces of the ester off the hormone, thereby releasing the active chemical (parenting steroid hormone) and allowing it to do its job (ultimately muscle building/tissue via protein synthesis, positive nitrogen balance and so on )... Now in the bloodstream which testosterone and blood born nutrients and substances circulate is recognized as the "medium". These enzymes circulate in the medium and directly affect the release of "active" hormone in the bloodstream by ester cleavage, thus exerting different expression per individuals, trigger a chute of cellular interaction at receptor sites that will differ from one to the next..

People need to remember that hormones are "chemical messengers" that rely messages to cells that display specific receptors for each hormone and respond to the signaling.. Depending on the ester and the individuals metabolization ratio the hormone can/may make changes directly to a cell, by changing the genes that are activated, or by making changes indirectly to a cell by stimulating other signaling pathways inside a specific cell group that is effected and effect other processes, thus this can "initiate" an intracellular cascade of events..

This is why the "same/singular hormone ester" effects people differently, because there's numerous ways a hormone can yield different effects.. So, depending on which transcription factors are present different genes will be transcribed in response...Thereby the reason some individuals tend to bloat and others do NOT, as some can run 250mgs of Test and begin to see signs of fluid retention or edema exert in the circular system, or particular regions of the body.. Yet, there's individuals that can utilize 1000mgs of Test and remain dry.. Of course diet permitting.. (but this article's about biosynthesis, and not diet)

Regards,
Vision
Click to expand...

I read it all. Great post

Max
#isohex
Lol
 
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