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What was your Favorite Products from EP/P.S.L. ?

Vision

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What was your Favorite Products from EP/P.S.L. ?

I'm always going to be a Test, Deca lover for EP, but I have to say that this past year was a game changer.. Stenbolone really caught my attention, and it was a fun little ride, stacked with var/tbol..

I'm looking forward to staking that once again this spring into summer... Test/Eq blend, with stenbolone and var/tbol combo..

What has been your favorties from P.S.L (EP) ?
 
What was your Favorite Products from EP/P.S.L. ?

I'm always going to be a Test, Deca lover for EP, but I have to say that this past year was a game changer.. Stenbolone really caught my attention, and it was a fun little ride, stacked with var/tbol..

I'm looking forward to staking that once again this spring into summer... Test/Eq blend, with stenbolone and var/tbol combo..

What has been your favorties from P.S.L (EP) ?
So far, it's been Trestolone. But the daily pinning did get old, and the scar tissue began to build up by the end of the cycle. However, I have NOT ever tried Stenbolone, Var, nor T-bol. I do have T-bol and var lined up, and on deck. The sten is something that I might consider if I don't have to go overseas for it....(hint hint)....
 
So far, it's been Trestolone. But the daily pinning did get old, and the scar tissue began to build up by the end of the cycle. However, I have NOT ever tried Stenbolone, Var, nor T-bol. I do have T-bol and var lined up, and on deck. The sten is something that I might consider if I don't have to go overseas for it....(hint hint)....

The sten is like a 2.0 version of mast.. stacked with var/tbol is simply great.. I'm with you on the daily pinning, it does get old especially if you have a tight schedule, man it can get old

I'll see what I can do for the local option! :D
 
Test E + EQ in first place.
I've bought more than 10 bottles and I'll never stop taking it.
400 mg/ml without any problem, not a pip, not a nuisance, nothing.
Excellent results and blood tests beyond the stars.



At second place, on equal merit, two great new discoveries (at least for me)
Stenbolone and M1T.
 
Anyone ever stack tren or deca with trest?
I've run 2 19-nors at the same time too so let's not go jumping down my throat. I like tren and deca together actually

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Anyone ever stack tren or deca with trest?
I've run 2 19-nors at the same time too so let's not go jumping down my throat. I like tren and deca together actually

Sent from my LG-SP320 using Tapatalk

Nothing wrong with tren deca mixed, if it works for you.. when tren ace would flatten me out I'd toss in NPP to assist with the muscle belly.. same thing, different ester.
 
Var seemed pretty legit. Clen cramped me so bad I had to cut it. Those stomach cramps are no joke.

Sent from my SM-G955U using Tapatalk
 
Anyone ever stack tren or deca with trest?
I've run 2 19-nors at the same time too so let's not go jumping down my throat. I like tren and deca together actually

Sent from my LG-SP320 using Tapatalk

Tren and trest are amazing together. A little tren goes a long way when stacked with trest.

Trest and stenbolone are my top two (not counting test) from PSL
 
Everything has been great company has been great to wife and I since we’ve came on board
 
Var seemed pretty legit. Clen cramped me so bad I had to cut it. Those stomach cramps are no joke.

Sent from my SM-G955U using Tapatalk
Clen makes me way too dehydrated as well. Cramps suck...

Sent from my LG-SP320 using Tapatalk
 
IML Gear Cream!
Tren and trest are amazing together. A little tren goes a long way when stacked with trest.

Trest and stenbolone are my top two (not counting test) from PSL
Everybody is loving on this sten...
Might be something to that...
What do you think about something like
250 test pw
100 trest ace eod
150 tren ace eod
1mg adex e4d (not prone to estrogenic side effects almost at all - I've run over 500mgs test pw with no AI)
Caber on hand of course...
I have some NPP, Mast and tren ace on hand so that will be the next go; however the cycle I threw up looks like it may be in the books. Maybe sub out the tren ace for sten?

Sent from my LG-SP320 using Tapatalk
 
.... I'm with you on the daily pinning, it does get old especially if you have a tight schedule, man it can get old
I usually begin getting scar tissue first in my delts. I can feel the needles going in tougher and tougher. And when I was on the Trestolone for 8 weeks, I was using eight different poke areas, ( Glutes, Triceps, quads, and delts). I'd like to add a couple more to make it ten injection sites. I've been thinking about adding biceps to the list, but the mere thought of poking biceps seems a little scary to me.
 
Everybody is loving on this sten...
Might be something to that...
What do you think about something like
250 test pw
100 trest ace eod
150 tren ace eod
1mg adex e4d (not prone to estrogenic side effects almost at all - I've run over 500mgs test pw with no AI)
Caber on hand of course...
I have some NPP, Mast and tren ace on hand so that will be the next go; however the cycle I threw up looks like it may be in the books. Maybe sub out the tren ace for sten?

Sent from my LG-SP320 using Tapatalk

That will be a very good cycle. Have you run trest before? The conversion rate is pretty high. I’m not prone to estrogen sides, but I did have to run my AI higher than normal because my e2 got up there and I was bloating. Just something to keep in mind. Run the sten with your next blast. Drop the mast. Npp/sten/tren will be phenomenal.
 
That will be a very good cycle. Have you run trest before? The conversion rate is pretty high. I’m not prone to estrogen sides, but I did have to run my AI higher than normal because my e2 got up there and I was bloating. Just something to keep in mind. Run the sten with your next blast. Drop the mast. Npp/sten/tren will be phenomenal.
Yeah never ran it. I've heard lf boob leakage on trest so I know it's no joke lmao

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Yeah never ran it. I've heard lf boob leakage on trest so I know it's no joke lmao

Sent from my LG-SP320 using Tapatalk

As long as you keep your estrogen in control, you’ll be fine. It’s not a drug to not take an ai with. The only downside I had with it was a little bloating. I could see it in my face.
 
Yeah never ran it. I've heard lf boob leakage on trest so I know it's no joke lmao

Sent from my LG-SP320 using Tapatalk
Let's put it this way...based on my one 8 week experience with Trestolone, if you wanted to deliberately grow breasts using an AAS, Trestolone would be the one to use. I had to use more Aromasin and Letrozole to stop the nipple pain, than I've ever used before on any other gear cycle I've ever done, and that includes VERY high dosages of Anadrol, Deca, Tren, and D-bol, (separately). NONE of those other AAS compounds ever gave me nipple pain as much, nor as often as 50mg/100mg ED of Trestolone did, regardless of the extreme dosages I've used with them in the past.
 
Today I happened to pick up a bottle of testosterone that I used long ago (I do not name the brand, so much is not present on the forum).


I compared it with what I currently use, EP-PSL.
The difference is clear and evident.
On the one hand there is a very thick and "oily" oil, on the other it seems almost water.


EP-PSL oil is really something phenomenal.
Now I can use 27G 3/4 needles, and I tried to make it pass without any difficulty even by a 30G (I do not use it because it's too short).



This to say that it's not so much the preferred product, in terms of the steroid it contains.
Each one is better with one than with another, but the thing they have in common and that makes them special is the oil used, the method to produce it, etc.


(even the packaging is perfect)
 
Let's put it this way...based on my one 8 week experience with Trestolone, if you wanted to deliberately grow breasts using an AAS, Trestolone would be the one to use. I had to use more Aromasin and Letrozole to stop the nipple pain, than I've ever used before on any other gear cycle I've ever done, and that includes VERY high dosages of Anadrol, Deca, Tren, and D-bol, (separately). NONE of those other AAS compounds ever gave me nipple pain as much, nor as often as 50mg/100mg ED of Trestolone did, regardless of the extreme dosages I've used with them in the past.
I think it has to do with the fact that it's a 19nor progestin AND it aromatizes into estrogen. That's a double attack on the nipples from estro AND prolactin. Most compounds are either one or the other. For example; tren and deca are 19nors which can cause prolactin issues in the breast but tren doesn't aromatize and deca aromatizes very slightly. Trest is a 19 nor so you have all the same issues with prolactin, plus the fact that it aromatizes like dbol. Certainly not for beginners and those who don't have their ancillaries dialed in.
 
Today I happened to pick up a bottle of testosterone that I used long ago (I do not name the brand, so much is not present on the forum).


I compared it with what I currently use, EP-PSL.
The difference is clear and evident.
On the one hand there is a very thick and "oily" oil, on the other it seems almost water.


EP-PSL oil is really something phenomenal.
Now I can use 27G 3/4 needles, and I tried to make it pass without any difficulty even by a 30G (I do not use it because it's too short).



This to say that it's not so much the preferred product, in terms of the steroid it contains.
Each one is better with one than with another, but the thing they have in common and that makes them special is the oil used, the method to produce it, etc.


(even the packaging is perfect)

logo_new.png

25ywqcw.jpg

Sensitive info

With Euro-Pharmacies Oil based compounds - Absolutely NO EO-OIL is used or other food derivatives for carrier delivery method of all parenting hormones due to allergic reactions with some carries.
EP's oil base products are smooth with less site irritation like most other carriers, while providing a more stable and longer shelf life of the suspended drugs.


EURO-PHARMACIES MANUFACTURING METHODS , SUPPLIES , SEMI PRODUCTS AND ALL OPERATING ELEMENTS ARE COMPLETELY/CONSTANTLY UP TO DATE WITH ALL THE LATEST GLOBAL STANDARDS.

1. Carrier Oils used in EP Injectable Products Line manufacturing processes are completely Pharmaceutical Grade.
100% Synthetic , Hypoallergenic and Completely Clear/Transparent in terms of Color properties.
(No tint- No impurity and Completely Infection/Contamination Proof)

A. Hormone Raw Materials batches applied in production processes Can Naturally differ in Color Tint Properties
Therefore it's sometimes possible that some particular EP Line Product Batches can differ in terms of color tint rate.(Due to 100% Clear - Transparent Carrier Oil Appearance Property)

2. Solely Pharmaceutical Grade Hormone Raw Materials used in EP Injectable Products Line manufacturing processes.
(Provided only by Official and Certified Pharmaceutical Manufacturers)

3. Solely Pharmaceutical Grade Hormone Raw Materials used in EP Oral Products Line manufacturing proccesses.
A. Professional and HIGHEST QUALITY Fillers applied to EP Oral Products Line. (ie. starch , PVP etc.)
More info

EP Line Crystal Clear|Pro. Carrier Oils Clarification

EP Line Crystal Clear|Pro. Carrier Oils Clarification
Original EP Line always used only Professional/Legit Pharma Grade Carrier Oils "Inside".
EP Line PG carrier Oils were SOLELY Created and Produced for "PG" AAS.
Of course products like Deca and all Trens are an exception clearly - due to their "essence" used ...
EP Line uses only Pharma Grade carrier oils (synthetic/hypoallergenic/smooth/most efficiently dissolved-merged with the "essence" - professional oil used only in Original Pharm. Grade Products)

No UGL on the market uses such Pro./Expensive Oils to our knowledge.Those carrier oils are available only to Legit/Registered Pharma Grade Manufacturers ... and to Us.
(We sincerely apologize if we EVENTUALLY missed anything on any UGL on the market about that matter)

EP Line carrier oils proved with 100% certainty:


1. Second to None - Highly smooth and Painless injection experience.
2. Body/System 100% Smooth & Effective applicability and response.
3. 100% Pharma Grade Feature of: dissolving/applying with the "essence" in the system/body right from the start.

EP Line "Essence" Origin and Quality matches those PG Carrier Oils Fully naturally.

What is Miglyol 812?


Miglyol 812 is also a clear neutral oil. Miglyol 812 is a liquid form of an MCT oil. An MCT oil is a medium-chain triglyceride. A medium-chain triglyceride is a man-made fat that that is partially manmade. It is identified by the way the carbon atoms are rearranged in the structure. Many people use MCTs, which are typically derived from coconut and palms oils and dairy fats, for improved diet and weight management.

According to the Cleveland Clinic, MCTs decrease metabolic syndrome, reduce abdominal obesity, and boost weight loss. Like miglyol 840, miglyol 812 is desired in toxicology and pharmaceuticals because it improves the solubility and absorption of injectables and oral compounds. Miglyol is soluble at 20 degrees Celsius. Additional properties of miglyol 812 are spreadability, penetration-promoting application, and high stability compared to natural oils. When used as a lotion, cream, or any other spreadable application, miglyol 812 is permeable and non-greasy; therefore, it does not obstruct the skin’s natural respiration.

Whether we use miglyol 812 or miglyol 840, our process filter the oil before use. While natural oils may seem like a better solution for your needs, it is important to consider miglyol compounds because they are manufactured in a way that ensures stability and purity. They can also be more cost effective.

PSL Team Supervisor,
Vision
 
I didn't know that any of PSL's gear uses MIG 840 oil. I also didn't know there is a MIG 812. As far as the MIG oils being "more cost effective" it's been my understanding that at least MIG 840 is more expensive than other carrier oils are.
 
I'm also curious as to whether all oils are now on Mig840 or just some are?
If just some are, how do we know which ones are in Mig840?

Sent from my LG-SP320 using Tapatalk
 
I think it has to do with the fact that it's a 19nor progestin AND it aromatizes into estrogen. That's a double attack on the nipples from estro AND prolactin. Most compounds are either one or the other. For example; tren and deca are 19nors which can cause prolactin issues in the breast but tren doesn't aromatize and deca aromatizes very slightly. Trest is a 19 nor so you have all the same issues with prolactin, plus the fact that it aromatizes like dbol. Certainly not for beginners and those who don't have their ancillaries dialed in.
You bring up a good point here^....I've used Trenbolone more than anything else, and despite using Cabergoline and/or Parlodel, I've ALWAYS experienced bloated ankles. However I'm presently 8 weeks in a Trenbolone cycle, and this time, no bloating at the ankles nor anywhere else, but in addition to the Trenbolone and Test mix, I'm also using 600mg per week of Masteron. Go figure. I cannot even explain this, nor can I draw any specific conclusion other than the Masteron making the difference.

So for my next Trestolone cycle, I plan on including Masteron to see how it works with that compound. I am not implying that Masteron will take the place of an aromataze inhibitor drug. Only that it has the potential to prevent the bloating
 
You bring up a good point here^....I've used Trenbolone more than anything else, and despite using Cabergoline and/or Parlodel, I've ALWAYS experienced bloated ankles. However I'm presently 8 weeks in a Trenbolone cycle, and this time, no bloating at the ankles nor anywhere else, but in addition to the Trenbolone and Test mix, I'm also using 600mg per week of Masteron. Go figure. I cannot even explain this, nor can I draw any specific conclusion other than the Masteron making the difference.

So for my next Trestolone cycle, I plan on including Masteron to see how it works with that compound.

Oh man,
Always run mast with tren. Is rule number one :)
 
I never knew that until now

Let's discuses Tren and one compound that can help assist with side effects that can be unbearable for most,especially anxiety..

Please allow me to illustrate one of shrouded and seldom discussed Drugs in the whole anabolic circuit, with one of the most underrated/pronounced effects ever, that somehow has failed to be discussed upon the masses...

"Proviron" Mesterolone or Msteron

Most of you that have ever took the breakfast of champions "Methandrostenolone", That's right, I'm talking about Dbol. What's the most apparent and conspicuous effects that takes place while taking Dbol? If you were about to say the "sense of well-being" than your correct. One of the most profound and desirable effects that we can have during a cycle..Now, how about after a cycle? Or for longer duration's? But we all know that many of us practice moderation with harsh orals,or I would hope,But, have no concerns with elevated toxicity here!

One of the greatest characteristics about Proviron that has been shrouded and seldomly discussed is it's "Antidepressant" properties. With this being said, when it was first developed it was widely utilized in treatments for Bi-polar,OCD and Anxiety. As we know that depression is basically a chemical imbalance that comes about through the "Signaling" issues between receptors. Proviron improves the quality of the "channels" that the cells use to communicate and interact. Thus, a similar effect with Dbol where it drastically improves the sense of well being in users. Much like Antidepressants, SSRI (Selective serotonin re-uptake inhibitor, and/or,SNRI (Serotonin-nor-epinephrinere-uptake inhibitor)

What I'm about to share is a double blind study that clearly shows undoubtedly astonishing results in the patients! An other great reason to consider this compound.
Why proviron is underestimated, the world may never know

Tren is the compound that's well known for having a love hate relationship with most users. Most will deem it a necessary evil. But, in fact it doesn't have to be classified as evil after all.

Allow me to intro some clinical studies that have been conducted with a compound most commonly known as Proviron-trade name (Mesterolone).This agent posses some amazing characteristics with Antidepressant properties, as well Anti-anxiety.
It works by also metabolizing and being recognized through the endocrine as (other) a neurosteroid,effectively functioning as a so-called proneurosteroid (testosterone is also recognized as one).. These steroids synthesized in the brain (Proviron especially) and have effects on brain function,In addition to their actions on neuronal membrane receptors,improving the quality of the channels that cells use to communicate and interact.

Proviron/or Masteron and Tren (Masteron can be utilized due to it's targeting similarities)
Proviron(mesterolone) will exert inhibitory actions on neurotransmission, acting as potent positive allosteric modulator of the GABA receptor (This is crucial concerning Tren-Insomnia as healthly function levels ofGABA will produce a stable sleep state/environment for rest) and possess, in no particular order, antidepressant,stress-reducing, feeling warm/fuzzy/rewarding,pro-social, anti-aggressive(huge consider tren sides),pro-sexual,sedative/pro-sleep,cognitive-memory improvement..The list goes on!

(Where does this apply with Tren? It can aid all the way around with individuals how are sensitive or not.From the social aspect,overwhelming sense of anxiety,lack of sleep,basically everything stated above that may apply with the usage of tren and the onset of its unwanted side)

In addition to this information, an individual can also utilized masteron (Drostanolonein) in conjunction with Proviron, running both concurrent may yield a great synergenic effect,each compound will compliment one an other.

Further more Proviron is a DHT derivative. DHT compounds assist with hardening of the physique, lack of water retention,increased sex drive..Hardening of the physique and lack of water retention go hand in hand. Proviron assists with this, The body recognizes proviron as a DHT,This causes a direct hardening affect on the muscle tissue (Like mast posses,but mast is much more stronger IMO) The increase in hardness comes from a reduction in free estrogen levels, because proviron has the ability to 'latch-on' to the estrogen binding enzymes,It competes so to speak for its position,it does this aggressively, thus decreasing water retention. Also the the lack of aromatization and the fact that the drug is prototypical androgen, causes a significant shift in the body’s estrogen/testosteroneratio.As proviron's atomic structure it is incapable of forming estrogen. It also has properties with AR's.. Increasing the AR expression, proviron/DHT uptake to further increase AR expression, repeating this process over and over ...
This allows other AAS compounds to appear to be amplified with there effects,assisting the compounds - (What does this mean?) It can be a master key so to speak, having multiple functions - It binds aggressively to the AR's and SHBG, thus it can/may increase the activity of other STEROIDS in the system) - This is an added bonus!

Learn more about Proviron here - PSL

Functions concerning the neurotransmitter/receptor and how it works:Below is a image illustrating the neurotransmitter/receptor and how it functions, also I will include some real actual studies conducted with proven results expressing the benefits of this compound (proviron)
Keep in mind that these doses may seem extreme,its been proven time and time again that such significant dosages are not needed to yield the effect. Merely a daily intake of 50-100 will suffice for almost anyone!
main-qimg-c1b39c3a7cbe266c827cf21bc88ce7e8


Citation
Database: PsycINFO
[ Journal Article ]
A comparison of the antidepressant effects of a synthetic androgen (mesterolone) and amitriptyline in depressed men.
Vogel, William; Klaiber, Edward L.; Broverman, Donald M.
Journal of Clinical Psychiatry, Vol 46(1), Jan 1985, 6-8.

Abstract

26 depressed male outpatients were randomly assigned to 14 wks of treatment with either mesterolone or amitriptyline in a double-blind parallel treatment design. Ss completed the Hamilton Rating Scale for Depression and a symptom checklist each week. Findings reveal that the drugs were equally effective in reducing depressive symptoms. Mesterolone produced significantly fewer adverse side effects than amitriptyline and did not produce hypomania or tachycardia, recognized side effects of amitriptyline. (10 ref) (PsycINFO Database Record (c) 2013 APA, all rights reserved)

Methods Find Exp Clin Pharmacol. 1984 Jun;6(6):331-7.
The effects of mesterolone, a male sex hormone in depressed patients (a double blind controlled study).
Itil TM, Michael ST, Shapiro DM, Itil KZ.

Abstract

Based on computer EEG (CEEG) profiles, in high doses, antidepressant properties of mesterolone, a synthetic androgen, were predicted. In a double-blind placebo controlled study, the clinical effects of 300-450 mg daily mesterolone were investigated in 52 relatively young (age range 26-53 years, mean 42.7 years) male depressed outpatients. During 6 weeks of mesterolone treatment, there was a significant improvement of depressive symptomatology. However, since an improvement was also established during the placebo treatment, no statistically appreciable difference in the therapeutic effects of mesterolone was established compared to placebo. Mesterolone treatment significantly decreased both plasma testosterone and protein bound testosterone levels. Patients with high testosterone levels prior to treatment seem to have had more benefit from mesterolone treatment than patients with low testosterone levels. The degree of improvement weakly correlated to the decrease of testosterone levels during mesterolone treatment.

Information confirming no HTPA shutdown/suppression during PCT

These are some research articles that may justify the use of low/moderate dose Proviron during PCT:

--------------------------------------------------------------------------------------------
AAKVAAG, A., and S. B. STROMME. "The effect of mesterolone administration to normal men on the pituitary-testicular function."Acta endocrinologica 77.2 (1974): 380-386.

ABSTRACT
Mesterolone (1***945;-methyl-5***945;-dihydrotestosterone) has been given to 10 normal men, age 24–27 years, and the effect on the plasma levels of ICSH, FSH and testosterone has been studied.No effect on the plasma levels of ICSH and FSH could be detected. After 4 weeks on 75 mg mesterolone per day a significant (P < 0.01) drop in the mean value for plasma testosterone level was observed, 5.2 to 4.0 ng/ml. After another 4 weeks on 150 mg mesterolone per day a further decrease to 3.5 ng/ml was found.During mesterolone administration the protein binding of testosterone in plasma was significantly reduced, and it appeared that the level of free (non-protein bound) testosterone in diluted plasma remained unchanged, 0.37 and 0.41 ng/ml, before and after mesterolone administration respectively.The results suggest that mesterolone given in doses of 75 and 150 mg/day to normal men does not suppress the pituitary ICSH production or the testicular testosterone production
--------------------------------------------------------------------------------------------

GORDON, R.D., THOMAS, M.J., POYNTING, J.M. and STOCKS, A.E. (1975), Effect of Mesterolone on Plasma L.H., F.S.H. and Testosterone. Andrologia, 7: 287–296. doi: 10.1111/j.1439-0272.1975.tb00942.x

Summary
It has been claimed that orally administered mesterolone, unlike l7a-methyl testo- sterone, does not suppress endogenous gonadotrophins and testesterone. To investi- gate this, both drugs were administered, in turn, to four normal men and plasma te- stosterone, L.H. and F.S.H. were measured serially. Mesterolone administration was associated in all four subjects with significant and similar falls in plasma testosterone, but significant suppression of gonadotrophins took place in only two of them. Any changes which occured were apparent by the end of the first week of therapy. Administration of half the dose of 17a-methyl testosterone to the same four subjects caused significant suppression of testosterone in each and suppression of one or both gonadotrophins in each.
In longer term studies in patients (5-30 months) involving serial measurements at intervals of one to two months, there was evidence of significant suppression of L.H. and F.S.H. by 17a-methyl testosterone, but not by mesterolone, which was clinically a less effective androgen.
--------------------------------------------------------------------------------------------

WANG, C., BURGER, H.G., de KRETSER, D.M., DULMANIS, A., HUDSON, B., KEOGH, E.J. and SUTHERS, M.B. (1974), Effect of Mesterolone on Serum FSH, LH and Plasma Testosterone in Normal Men. Andrologia, 6: 111–117. doi: 10.1111/j.1439-0272.1974.tb01604.x

Summary
To determine whether the claim that mesterolone, an orally active androgen, does not cause suppression of gonadotrophin secretion, two groups of five normal men were treated with 100 and 200 mg. daily respectively for 7 days. Serial measurements of serum FSH, LH and plasma testosterone were made on samples taken at 15 minute intervals over 2 hr both before and during treatment. Modest falls in FSH, LH and testosterone levels were observed in both groups, the percentage suppression being 21% and 18% for FSH, 19% and 15% for LH and 9% and 8% for testosterone at the lower and higher dosage levels respectively.
--------------------------------------------------------------------------------------------

200mg is a far greater dose than I would employ during PCT (50mgs is ideal). From these studies and other articles we see have read, it's clear that there is almost minimal/no influence on the HTPA, any effect would be absolute minimal and negated by other appropriate compounds used during that period (HCG, Aromasin, Nolvadex and HGH)...

Team PSL supervisor
Vision
 
MasteronE can be used in place of provirion

The truth about mesterolone (proviron) :::Buy it here:::

PROVcleaned1.jpg


Here is some info in regards to proviron, it includes the benefit used with Test, tren, and assistance with low test levels and/or even depression....

What makes Priviron extremely beneficial in cycles/blast?

There's a few plausible explanations for this..

Proviron being a DHT in way it can/will lower SHBG levels which hinders estrogen formulation, thus providing the muscles with that "hard,dry appearance, less water retention"..Appearance so to speak, but there's more truth to this as I will explain below..

Proviron/mast work with a few different pathways, on the inside and outside of the AR's..Like mentioned in the initial post above, Proviron and Mast have been shown to increase the amount of free flowing test/bioavailability, greater blood plasma with Free test rather then bound, at the same time assisting test to stay in it's instructed pathways.. As you may know, Test has 3 functions.. 1)it's main course of action,being a main sex hormone/testosterone..2) Converting into estro, but lower SHBG hinders this in the presence of proviron..3) Test can convert into a MORE potent androgen such as DHT, and we know the functions of DHT as it tend to have a greater affinity for proteins over Test..

Testosterone alone has many functions that's often over looked when it concerns someone attempting to achive a harder, leaner look, in lieu people focus on other compounds when in fact Test in conjunction with proviron has a very pivotal role..

How does one achieve more definition,harder,leaner,more dry?
Now in the continuous supply of proviron or mast other AAS activities are almost amplified or undisturbed, allowing a great instance of protein synthesis..

(FYI; Androgen receptors are found throughout cellular groups, as well as FAT and muscle cell/groups, now we know that they initiate a response on AR's in muscle cells to promote size/growth, at the same given time they will have a cascade of effects on other cells and AR's found therein fat cells inducing activity/burning, thus the addition of proviron assist with AAS in these targeted tissue groups...Higher/more potent the androgen binds to the androgen receptors, the greater the lipolytic response will be on adipose tissue (brown or white)

Now lets also take into great consideration AR upregulation with the presence of androgens (more free flowing test), more AR sites throughout targeting tissue..,There's a vast amount of activity in which a complex interplay between activation and inactivation mechanisms and signaling between cell groups, what People need to remember that hormones are "chemical messengers" that rely messages to cells that display specific receptors for each hormone and respond to the signaling..Depending on the compounds and the individuals metabolization ratio the hormone can/may make changes directly to a cell, by changing the genes that are activated, or by making changes indirectly to a cell by stimulating other signaling pathways inside a specific cell group that is effected and effect other processes, thus this can "initiate" an intracellular cascade of events.. So, the notion that fat loss is NOT presence with proviron or mast, and to mitigate that AAS don't posses any fat loss properties is absurd...Should someone dial down their body fat to see greater results? This would be wise, but it's crucial..

IMO even a couch potato can see results with AAS, proviron, test, ext.. Now whether that lazy f*ck is deserving of it, that's a whole different topic!

So, in a nut shell.. Less presence of water retention, great affinity with protein vai muscle tissue, recovery, less estro related effects in specific targeting tissue, lower SHBG, more bio active test and presence from other androgens in or around AR's and other mediated tissue/muscle cells and other cellular groups and internal switches, improved pathways and other things etc etc etc..

Now mentions in the above abscracts about SHBG - SHBG is in fact the MASTER REGULATOR for Testosterone and Estrogen, having shifts within the static system and imbalances are often precipitated by SHBG abnormalities whether on or off AAS..

You don'there much mention about SHBG by come phtisicians while testing for T-serum level and I can explain why, That's because most don't even test for it, true story.. In fact most endo's go by the staple text book when it concerns therapeutic levels (by way of TRT)..But, we're not talking about therapeutic levels here, now when taking above theraputic levels there's enough evidence to support the importance of SHBG levels and the vital role of free test..

Free test is a VERY important number to monitor and maintain, as research has shown again and again, that free test is the one that actually works on skeletal tissue, in liue of a reserve bank..Total test matters without a doubt, because of the reserve that free test can built from..Bound Test to albumin may/can be bonded very loosely (depending in individuals ) and it will be ready to be available to do work when needed..

For those that may question the very importance of SHBG and suggesting SHBG means nothing while running Test, what about individuals that are prone to estrogen related sides? Higher SHBG may result in less active test, and more active estro in sensitive users..

Most endos only check for total test, that is all, unless a patient isn't reaching particular numbers within a range on a specific dosage, then they will order a panel for free test serum and SHBG looking for abnormalities..Keep in mind that even a great total test score can yield potentially false readings of what's actually taking place..Text book protocol can be very misleading.. For great reason I have such biased feelings towards Proviron..This read may summarizes on the pronounced properties of this underrated compound..


With this said lets discuss how it can assist with depression,aggression and/or anxiety,or other sides when on cycle/blast when utilizing Trenbolone, or at times other compounds and the same time "improving there performance/mechanisms..

Let's discuses Tren and one compound that can help assist with side effects that can be unbearable for most,especially anxiety..So lets talk about this shall we? There's more info added in regards to proviron being utilized with Test or other AAS, but the opening will cover the "Benefits while using Tren/Proviron combo"

Please allow me to illustrate one of the most shrouded and seldom discussed Drugs in the whole anabolic circuit, with one of the most underrated/pronounced effects ever, that somehow has failed to be discussed upon the masses...

"Proviron" Mesterolone
(Masteron also applies as Proviron and mast can share the same pathways and expressions)

Most of you that have ever took the breakfast of champions "Methandrostenolone", That's right, I'm talking about Dbol. What's the most apparent and conspicuous effects that takes place while taking Dbol? If you were about to say the "sense of well-being" than your correct. One of the most profound and desirable effects that we can have during a cycle..Now, how about after a cycle? Or for longer duration's? But we all know that many of us practice moderation with harsh orals,or I would hope,But, have no concerns with elevated toxicity here!

One of the greatest characteristics about Proviron that has been shrouded and seldomly discussed is it's "Antidepressant" properties. With this being said, when it was first developed it was widely utilized in treatments for Bi-polar,OCD and Anxiety. As we know that depression is basically a chemical imbalance that comes about through the "Signaling" issues between receptors. Proviron improves the quality of the "channels" that the cells use to communicate and interact. Thus, a similar effect with Dbol where it drastically improves the sense of well being in users. Much like Antidepressants, SSRI (Selective serotonin re-uptake inhibitor, and/or,SNRI (Serotonin-nor-epinephrinere-uptake inhibitor)

What I'm about to share is a double blind study that clearly shows undoubtedly astonishing results in the patients! An other great reason to consider this compound.
Why proviron is underestimated, the world may never know

Tren is the compound that's well known for having a love hate relationship with most users. Most will deem it a necessary evil. But, in fact it doesn't have to be classified as evil after all..

Allow me to intro some clinical studies that have been conducted with a compound most commonly known as Proviron-trade name (Mesterolone).This agent posses some amazing characteristics with Antidepressant properties, as well Anti-anxiety.
It works by also metabolizing and being recognized through the endocrine as (other) a neurosteroid,effectively functioning as a so-called proneurosteroid (testosterone is also recognized as one).. These agents synthesized in the brain (Proviron especially) and have effects on brain function,In addition to their actions on neuronal membrane receptors,improving the quality of the channels that cells use to communicate and interact.

Proviron/or Masteron and Tren (Masteron can be utilized due to it's targeting similarities)
Proviron(mesterolone) will exert inhibitory actions on neurotransmission, acting as potent positive allosteric modulator of the GABA receptor (This is crucial concerning Tren-Insomnia as healthly function levels of GABA will produce a stable sleep state/environment for rest) and possess, in no particular order, antidepressant,stress-reducing, feeling warm/fuzzy/rewarding,pro-social, anti-aggressive(huge consider tren sides),pro-sexual,sedative/pro-sleep,cognitive-memory improvement..The list goes on!

(Where does this apply with Tren? It can aid all the way around with individuals how are sensitive or not.From the social aspect,overwhelming sense of anxiety,lack of sleep,basically everything stated above that may apply with the usage of tren and the onset of its unwanted side)

In addition to this information, an individual can also utilized masteron (Drostanolonein) in conjunction with Proviron, running both concurrent may yield a great synergenic effect,each compound will compliment one an other.

Below is a image illustrating the neurotransmitter/receptor and how it functions, also I will include some real actual studies conducted with proven results expressing the benefits of this compound (proviron)
Keep in mind that these doses may seem extreme,its been proven time and time again that such significant dosages are not needed to yield the effect. Merely a daily intake of 50-100 will suffice for almost anyone!
350px-Synapse_Illustration2_tweaked.svg.png

PROV2cleaned2.jpg


Further more Proviron is a DHT derivative. DHT compounds assist with hardening of the physique, lack of water retention,increased sex drive..Hardening of the physique and lack of water retention go hand in hand. Proviron assists with this, The body recognizes proviron as a DHT,This causes a direct hardening affect on the muscle tissue (Like mast posses,but mast is much more stronger IMO) The increase in hardness comes from a reduction in free estrogen levels, because proviron has the ability to 'latch-on' to the estrogen binding enzymes,It competes so to speak for its position,it does this aggressively, thus decreasing water retention. Also the the lack of aromatization and the fact that the drug is prototypical androgen, causes a significant shift in the body***8217;s estrogen/testosteroneratio.As proviron's atomic structure it is incapable of forming estrogen. It also has properties with AR's.. Increasing the AR expression, proviron/DHT uptake to further increase AR expression, repeating this process over and over ...
This allows other AAS compounds to appear to be amplified with there effects,assisting the compounds - (What does this mean?) It can be a master key so to speak, having multiple functions - It binds aggressively to the AR's and SHBG, thus it can/may increase the activity of other AAS/Androgens in the system) - This is an added bonus!

Functions concerning the neurotransmitter/receptor and how it works:Below is a image illustrating the neurotransmitter/receptor and how it functions, also I will include some real actual studies conducted with proven results expressing the benefits of this compound (proviron)
Keep in mind that these doses may seem extreme,its been proven time and time again that such significant dosages are not needed to yield the effect. Merely a daily intake of 50-100 will suffice for almost anyone!

Citation
Database: PsycINFO
[ Journal Article ]
A comparison of the antidepressant effects of a synthetic androgen (mesterolone) and amitriptyline in depressed men.
Vogel, William; Klaiber, Edward L.; Broverman, Donald M.
Journal of Clinical Psychiatry, Vol 46(1), Jan 1985, 6-8.

Abstract

26 depressed male outpatients were randomly assigned to 14 wks of treatment with either mesterolone or amitriptyline in a double-blind parallel treatment design. Ss completed the Hamilton Rating Scale for Depression and a symptom checklist each week. Findings reveal that the drugs were equally effective in reducing depressive symptoms. Mesterolone produced significantly fewer adverse side effects than amitriptyline and did not produce hypomania or tachycardia, recognized side effects of amitriptyline. (10 ref) (PsycINFO Database Record (c) 2013 APA, all rights reserved)

Methods Find Exp Clin Pharmacol. 1984 Jun;6(6):331-7.
The effects of mesterolone, a male sex hormone in depressed patients (a double blind controlled study).
Itil TM, Michael ST, Shapiro DM, Itil KZ.
Abstract
Based on computer EEG (CEEG) profiles, in high doses, antidepressant properties of mesterolone, a synthetic androgen, were predicted. In a double-blind placebo controlled study, the clinical effects of 300-450 mg daily mesterolone were investigated in 52 relatively young (age range 26-53 years, mean 42.7 years) male depressed outpatients. During 6 weeks of mesterolone treatment, there was a significant improvement of depressive symptomatology. However, since an improvement was also established during the placebo treatment, no statistically appreciable difference in the therapeutic effects of mesterolone was established compared to placebo. Mesterolone treatment significantly decreased both plasma testosterone and protein bound testosterone levels. Patients with high testosterone levels prior to treatment seem to have had more benefit from mesterolone treatment than patients with low testosterone levels. The degree of improvement weakly correlated to the decrease of testosterone levels during mesterolone treatment.

Information confirming no HTPA shutdown/suppression during PCT

These are some research articles that may justify the use of low/moderate dose Proviron during PCT:

--------------------------------------------------------------------------------------------
AAKVAAG, A., and S. B. STROMME. "The effect of mesterolone administration to normal men on the pituitary-testicular function."Acta endocrinologica 77.2 (1974): 380-386.

ABSTRACT
Mesterolone (1***945;-methyl-5***945;-dihydrotestosterone) has been given to 10 normal men, age 24***8211;27 years, and the effect on the plasma levels of ICSH, FSH and testosterone has been studied.No effect on the plasma levels of ICSH and FSH could be detected. After 4 weeks on 75 mg mesterolone per day a significant (P < 0.01) drop in the mean value for plasma testosterone level was observed, 5.2 to 4.0 ng/ml. After another 4 weeks on 150 mg mesterolone per day a further decrease to 3.5 ng/ml was found.During mesterolone administration the protein binding of testosterone in plasma was significantly reduced, and it appeared that the level of free (non-protein bound) testosterone in diluted plasma remained unchanged, 0.37 and 0.41 ng/ml, before and after mesterolone administration respectively.The results suggest that mesterolone given in doses of 75 and 150 mg/day to normal men does not suppress the pituitary ICSH production or the testicular testosterone production
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GORDON, R.D., THOMAS, M.J., POYNTING, J.M. and STOCKS, A.E. (1975), Effect of Mesterolone on Plasma L.H., F.S.H. and Testosterone. Andrologia, 7: 287***8211;296. doi: 10.1111/j.1439-0272.1975.tb00942.x

Summary
It has been claimed that orally administered mesterolone, unlike l7a-methyl testo- sterone, does not suppress endogenous gonadotrophins and testesterone. To investi- gate this, both drugs were administered, in turn, to four normal men and plasma te- stosterone, L.H. and F.S.H. were measured serially. Mesterolone administration was associated in all four subjects with significant and similar falls in plasma testosterone, but significant suppression of gonadotrophins took place in only two of them. Any changes which occured were apparent by the end of the first week of therapy. Administration of half the dose of 17a-methyl testosterone to the same four subjects caused significant suppression of testosterone in each and suppression of one or both gonadotrophins in each.
In longer term studies in patients (5-30 months) involving serial measurements at intervals of one to two months, there was evidence of significant suppression of L.H. and F.S.H. by 17a-methyl testosterone, but not by mesterolone, which was clinically a less effective androgen.
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WANG, C., BURGER, H.G., de KRETSER, D.M., DULMANIS, A., HUDSON, B., KEOGH, E.J. and SUTHERS, M.B. (1974), Effect of Mesterolone on Serum FSH, LH and Plasma Testosterone in Normal Men. Andrologia, 6: 111***8211;117. doi: 10.1111/j.1439-0272.1974.tb01604.x

Summary
To determine whether the claim that mesterolone, an orally active androgen, does not cause suppression of gonadotrophin secretion, two groups of five normal men were treated with 100 and 200 mg. daily respectively for 7 days. Serial measurements of serum FSH, LH and plasma testosterone were made on samples taken at 15 minute intervals over 2 hr both before and during treatment. Modest falls in FSH, LH and testosterone levels were observed in both groups, the percentage suppression being 21% and 18% for FSH, 19% and 15% for LH and 9% and 8% for testosterone at the lower and higher dosage levels respectively.
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200mg is a far greater dose than I would deploy during PCT (50mgs is ideal). From these studies and other articles we see have read, it's clear that there is almost minimal/no influence on the HTPA, any effect would be absolute minimal and negated by other appropriate compounds used during that period (HCG, Aromasin, Nolvadex and HGH)...

Vision

-----------------------

I'd also like to add some stellar information about "provirion" and its amazing proprieties..

Provirion won't suppress LH or FSH and will even provide your body with more free testosterone due to the fact that it binds "AGGRESSIVELY" to SHBG (lowering levels)....With this said it's not suppressive like most steroids, and your FREE T can greatly increase by allowing bound test to act in its original course of action..in lieu of being bound (freeing up bound test)

Proviron will assist in a blast/cycle
It's already known that only a very small amount of Testosterone exists as free testosterone. Free test is testosterone that capable of binding to the Androgen Receptor, which is where all the rest of the magic happens, and allows for the following benefits:
-Enhanced growth factor activity (e.g. GH, IGF-1, etc.)
-Enhanced activation of myogenic stem cells (i.e. satellite cells)
-Enhanced myonuclear number (to maintain nuclear to cytoplasmic ratio)
-Enhanced protein synthesis
-New myofiber formation

Testosterone binds at around 45% to what is known as Sex Hormone Binding Globulin (SHBG), and about another 53% binds to proteins (albumin). The rest exists in a free state (about 2% if you did your math). Different variations of steroids also differ in the way in which they bind to proteins.
If one could unbind testosterone from SHBG by even a small percentage, it could make a big difference in the way that testosterone or other AAS exert their anabolic effects. Studies show that when testosterone is unbound from SHBG the free test does in fact exert greater effects than total T. As the following studies support:

-
Demisch K, and Nickelsen T. Distribution of testosterone in plasma proteins during replacement therapy with testosterone enanthatein patients suffering from hypogonadism Andrologia 1983;15 Spec No:536-41.

Gandar R. Interpretation of the blood level of a steroid Rev Fr Gynecol Obstet 1985 Aug-Sep;80(8-9):635-40.

Legrand E., et al. Osteoporosis in men: a potential role for the sex hormone binding globulin Bone 2001 Jul;29(1):90-5.

Longcope C., et al. Diet and sex hormone-binding globulin. J Clin Endocrinol Metab 2000 Jan;85(1):293-296.

Valero-Politi J, and Fuentes-Arderiu X. Within- and between-subject biological variations of follitropin, lutropin, testosterone, and sex-hormone-binding globulin in men. Clin Chem 1993 Aug;39(8):1723-1725.

Proviron(1-Methyl Dihydrotestosterone) has been shown to bind with SHBG much more readily than test.

Relative binding affinity of anabolic-androgenic steroids: comparison of the binding to the androgen receptors in skeletal muscle and in prostate, as well as to sex hormone-binding globulin.

Saartok T, Dahlberg E, Gustafsson JA.

It is unclear whether anabolic steroids act on skeletal muscle via the androgen receptor (AR) in this tissue, or whether there is a separate anabolic receptor. When several anabolic steroids were tested as competitors for the binding of [3H]methyltrienolone (MT; 17 beta-hydroxy-17 alpha-methyl-4,9,11-estratrien-3-one) to the AR in rat and rabbit skeletal muscle and rat prostate, respectively, MT itself was the most efficient competitor. 1 alpha-Methyl-5 alpha-dihydrotestosterone (1 alpha-methyl-DHT; mesterolone) bound most avidly to sex hormone-binding globulin (SHBG) [relative binding affinity (RBA) about 4 times that of DHT]. Some anabolic-androgenic steroids bound strongly to the AR in skeletal muscle and prostate [ RBAs relative to that of MT: MT greater than 19-nortestosterone ( NorT ; nandrolone ) greater than methenolone (17 beta-hydroxy-1-methyl-5 alpha-androst-1-en-3-one) greater than testosterone (T) greater than 1 alpha-methyl-DHT]. In other cases, AR binding was weak (RBA values less than 0.05):stanozolol(17 alpha-methyl-5 alpha- androstano [3,2-c]pyrazol-17 beta-ol), methanedienone (17 beta-hydroxy-17 alpha-methyl-1,4-androstadien-3-one), and fluoxymesterolone (9 alpha-fluoro-11 beta-hydroxy-17 alpha-methyl-T). Other compounds had RBAs too low to be determined (e.g. oxymetholone (17 beta-hydroxy-2-hydroxymethylene-17 alpha-methyl-5 alpha-androstan-3-one) and ethylestrenol (17 alpha-ethyl-4- estren -17 beta-ol). The competition pattern was similar in muscle and prostate, except for a higher RBA of DHT in the prostate. The low RBA of DHT in muscle was probably due to the previously reported rapid reduction of its 3-keto function to metabolites, which did not bind to the AR [5 alpha-androstane-3 alpha, 17 beta-diol and its 3 beta-isomer (3 alpha- and 3 beta-adiol, respectively)]. Some anabolic-androgenic steroids (only a few synthetic) bound to SHBG (1 alpha-methyl-DHT much greater than DHT greater than T greater than 3 beta-adiol greater than 3 alpha-adiol = 17 alpha-methyl-T greater than methenolone greater than methanedienone greater than stanozolol). The ratio of the RBA in rat muscle to that in the prostate (an estimate of the myotrophic potency of the compounds) was close to unity, varying only between about 0.4 and 1.7 in most cases.(ABSTRACT TRUNCATED AT 400 WORDS)

Skalba P, Korfanty A, Mroczka W, Wojtowicz M. Related Articles
[Changes of SHBG concentrations in postmenopausal women]
Ginekol Pol. 2001 Dec;72(12A):1388-92. Polish.

Variations of sex hormone-binding globulin in thyroid dysfunction.

Brenta G, Schnitman M, Gurfinkiel M, Damilano S, Pierini A, Sinay I, Pisarev MA.

Department of Endocrinology and Metabolism, French Hospital, Buenos Aires,
Argentina.brenta@cnea.gov.ar


With the aim of understanding the variations of the levels of sex hormone-binding globulin (SHBG) in thyroid dysfunction, we studied the influence of factors that also modify SHBG, such as menopausal status, age, and body mass index (BMI) in women with hypothyroidism and hyperthyroidism, both overt and subclinical. Statistical analysis was performed by means of analysis of variance (ANOVA), stepwise multiple regression, and partial correlation. The ANOVA showed a significant statistical difference among the means of SHBG of all groups (p<0.01). The difference was due to the group that included hyperthyroid women. Multiple regression analysis showed that the main factors influencing SHBG were BMI and age, except for the hyperthyroid group, where the most important independent variables were triiodothyronine (T3) and thyroxine (T4). Partial correlation controlling the effect of BMI and age showed no association between SHBG and the other variables in all groups except for the subclinical hyperthyroid and hyperthyroid, where we found a significant association between SHBG and T4 and T3. The premenopausal or postmenopausal status did not modify SHBG levels. When the patients are taken as a whole, BMI, age, T4, and T3 all have an association with SHBG levels according to the multiple regression analysis.

Determinants of sex hormone-binding globulin blood concentrations in premenopausal and postmenopausal women with different estrogen status. Virgilio-Menopause-Health Group.

Pasquali R, Vicennati V, Bertazzo D, Casimirri F, Pascal G, Tortelli O, Labate AM.

Department of Internal Medicine and Gastroenterology, University of Bologna, Italy

Just a quote: 2) SHBG values are correlated positively with estradiol and negatively withinsulin and testosterone concentrations, but the predictive value of these variabiles on SHBG appears to be different in premenopause and postmenopause;

Here is a fun little fact: the level of SHBG can also be influenced by other factors. There is a direct relationship between the level of estrogen and thyroid hormones and the level of SHBG. Estrogen goes up, SHBG goes up. Estrogen goes down SHBG goes down. Same for Thyroid hormones triiodothyronine (T3) and thyroxine (T4). Also, there is a relationship with diet and insulin, but that is something I will save for later. Higher androgen levels due to AS administration has been shown to considerably lower levels of SHBG as well. The ASWinstrol (stanozolol) was shown in a 1989 study to lower levels of SHBG by 50% after oral administration.


Sex hormone-binding globulin response to the anabolic steroid stanozolol: evidence for its suitability as a biological androgen sensitivity test.

Sinnecker G, Kohler S.

Department of Pediatrics, University of Hamburg, West Germany.


Both the androgen-induced decline in serum sex hormone-binding globulin (SHBG) levels during puberty and the anabolic effect of exogenous testosterone are absent in patients with androgen insensitivity (testicular feminization). To determine whether the androgen-induced decline in serum SHBG could be used as a test of androgen sensitivity, we studied the effect of the anabolic-androgenic steroid stanozolol (17 beta-hydroxy-17 alpha-methyl-5 alpha-androstano-[3,2-c]pyrazol) on serum SHBG in 25 control subjects, 3 patients with complete androgen insensitivity, and 4 patients with partial androgen insensitivity. Stanozolol was administered orally for 3 days (0.2 mg/kg.day); blood samples were taken before and 5, 6, 7, and 8 days after the beginning of the test for measurements of serum SHBG. The lowest value (i.e. the peak response) in each subject was used as the measure of the response to stanozolol. In the control subjects the mean nadir serum SHBG level was 51.6 +/- 5.9% (+/- SD) of the initial value (P less than 0.001). In the 4 patients with partial androgen insensitivity the nadir serum SHBG ranged from 73-89%, and in the 3 patients with complete androgen insensitivity it ranged from 93-97% of the initial value. Thus, the decrease in serum SHBG after short term administration of stanozolol reflects androgen responsiveness and, thus, may be used to differentiate patients with androgen insensitivity syndromes from those with other causes of male pseudohermaphroditism.

Proviron could provide the mechanisms to increase the value of other AS... Proviron would work because by binding to SHBG, it leaves hormone in a free state to bind to the AR. Proviron is a terrible Anabolic, but its affinity for SHBG would essentially displace other steroids from binding to SHBG...Provirion would work to reduce the overall amount of SHBG, thereby having the effect of freeing up hormone to bind to the AR.

(Click here to order your Proviron TODAY)

 
The truth about mesterolone (proviron) :::Buy it here:::

PROVcleaned1.jpg


Here is some info in regards to proviron, it includes the benefit used with Test, tren, and assistance with low test levels and/or even depression....

What makes Priviron extremely beneficial in cycles/blast?

There's a few plausible explanations for this..

Proviron being a DHT in way it can/will lower SHBG levels which hinders estrogen formulation, thus providing the muscles with that "hard,dry appearance, less water retention"..Appearance so to speak, but there's more truth to this as I will explain below..

Proviron/mast work with a few different pathways, on the inside and outside of the AR's..Like mentioned in the initial post above, Proviron and Mast have been shown to increase the amount of free flowing test/bioavailability, greater blood plasma with Free test rather then bound, at the same time assisting test to stay in it's instructed pathways.. As you may know, Test has 3 functions.. 1)it's main course of action,being a main sex hormone/testosterone..2) Converting into estro, but lower SHBG hinders this in the presence of proviron..3) Test can convert into a MORE potent androgen such as DHT, and we know the functions of DHT as it tend to have a greater affinity for proteins over Test..

Testosterone alone has many functions that's often over looked when it concerns someone attempting to achive a harder, leaner look, in lieu people focus on other compounds when in fact Test in conjunction with proviron has a very pivotal role..

How does one achieve more definition,harder,leaner,more dry?
Now in the continuous supply of proviron or mast other AAS activities are almost amplified or undisturbed, allowing a great instance of protein synthesis..

(FYI; Androgen receptors are found throughout cellular groups, as well as FAT and muscle cell/groups, now we know that they initiate a response on AR's in muscle cells to promote size/growth, at the same given time they will have a cascade of effects on other cells and AR's found therein fat cells inducing activity/burning, thus the addition of proviron assist with AAS in these targeted tissue groups...Higher/more potent the androgen binds to the androgen receptors, the greater the lipolytic response will be on adipose tissue (brown or white)

Now lets also take into great consideration AR upregulation with the presence of androgens (more free flowing test), more AR sites throughout targeting tissue..,There's a vast amount of activity in which a complex interplay between activation and inactivation mechanisms and signaling between cell groups, what People need to remember that hormones are "chemical messengers" that rely messages to cells that display specific receptors for each hormone and respond to the signaling..Depending on the compounds and the individuals metabolization ratio the hormone can/may make changes directly to a cell, by changing the genes that are activated, or by making changes indirectly to a cell by stimulating other signaling pathways inside a specific cell group that is effected and effect other processes, thus this can "initiate" an intracellular cascade of events.. So, the notion that fat loss is NOT presence with proviron or mast, and to mitigate that AAS don't posses any fat loss properties is absurd...Should someone dial down their body fat to see greater results? This would be wise, but it's crucial..

IMO even a couch potato can see results with AAS, proviron, test, ext.. Now whether that lazy f*ck is deserving of it, that's a whole different topic!

So, in a nut shell.. Less presence of water retention, great affinity with protein vai muscle tissue, recovery, less estro related effects in specific targeting tissue, lower SHBG, more bio active test and presence from other androgens in or around AR's and other mediated tissue/muscle cells and other cellular groups and internal switches, improved pathways and other things etc etc etc..

Now mentions in the above abscracts about SHBG - SHBG is in fact the MASTER REGULATOR for Testosterone and Estrogen, having shifts within the static system and imbalances are often precipitated by SHBG abnormalities whether on or off AAS..

You don'there much mention about SHBG by come phtisicians while testing for T-serum level and I can explain why, That's because most don't even test for it, true story.. In fact most endo's go by the staple text book when it concerns therapeutic levels (by way of TRT)..But, we're not talking about therapeutic levels here, now when taking above theraputic levels there's enough evidence to support the importance of SHBG levels and the vital role of free test..

Free test is a VERY important number to monitor and maintain, as research has shown again and again, that free test is the one that actually works on skeletal tissue, in liue of a reserve bank..Total test matters without a doubt, because of the reserve that free test can built from..Bound Test to albumin may/can be bonded very loosely (depending in individuals ) and it will be ready to be available to do work when needed..

For those that may question the very importance of SHBG and suggesting SHBG means nothing while running Test, what about individuals that are prone to estrogen related sides? Higher SHBG may result in less active test, and more active estro in sensitive users..

Most endos only check for total test, that is all, unless a patient isn't reaching particular numbers within a range on a specific dosage, then they will order a panel for free test serum and SHBG looking for abnormalities..Keep in mind that even a great total test score can yield potentially false readings of what's actually taking place..Text book protocol can be very misleading.. For great reason I have such biased feelings towards Proviron..This read may summarizes on the pronounced properties of this underrated compound..


With this said lets discuss how it can assist with depression,aggression and/or anxiety,or other sides when on cycle/blast when utilizing Trenbolone, or at times other compounds and the same time "improving there performance/mechanisms..

Let's discuses Tren and one compound that can help assist with side effects that can be unbearable for most,especially anxiety..So lets talk about this shall we? There's more info added in regards to proviron being utilized with Test or other AAS, but the opening will cover the "Benefits while using Tren/Proviron combo"

Please allow me to illustrate one of the most shrouded and seldom discussed Drugs in the whole anabolic circuit, with one of the most underrated/pronounced effects ever, that somehow has failed to be discussed upon the masses...

"Proviron" Mesterolone
(Masteron also applies as Proviron and mast can share the same pathways and expressions)

Most of you that have ever took the breakfast of champions "Methandrostenolone", That's right, I'm talking about Dbol. What's the most apparent and conspicuous effects that takes place while taking Dbol? If you were about to say the "sense of well-being" than your correct. One of the most profound and desirable effects that we can have during a cycle..Now, how about after a cycle? Or for longer duration's? But we all know that many of us practice moderation with harsh orals,or I would hope,But, have no concerns with elevated toxicity here!

One of the greatest characteristics about Proviron that has been shrouded and seldomly discussed is it's "Antidepressant" properties. With this being said, when it was first developed it was widely utilized in treatments for Bi-polar,OCD and Anxiety. As we know that depression is basically a chemical imbalance that comes about through the "Signaling" issues between receptors. Proviron improves the quality of the "channels" that the cells use to communicate and interact. Thus, a similar effect with Dbol where it drastically improves the sense of well being in users. Much like Antidepressants, SSRI (Selective serotonin re-uptake inhibitor, and/or,SNRI (Serotonin-nor-epinephrinere-uptake inhibitor)

What I'm about to share is a double blind study that clearly shows undoubtedly astonishing results in the patients! An other great reason to consider this compound.
Why proviron is underestimated, the world may never know

Tren is the compound that's well known for having a love hate relationship with most users. Most will deem it a necessary evil. But, in fact it doesn't have to be classified as evil after all..

Allow me to intro some clinical studies that have been conducted with a compound most commonly known as Proviron-trade name (Mesterolone).This agent posses some amazing characteristics with Antidepressant properties, as well Anti-anxiety.
It works by also metabolizing and being recognized through the endocrine as (other) a neurosteroid,effectively functioning as a so-called proneurosteroid (testosterone is also recognized as one).. These agents synthesized in the brain (Proviron especially) and have effects on brain function,In addition to their actions on neuronal membrane receptors,improving the quality of the channels that cells use to communicate and interact.

Proviron/or Masteron and Tren (Masteron can be utilized due to it's targeting similarities)
Proviron(mesterolone) will exert inhibitory actions on neurotransmission, acting as potent positive allosteric modulator of the GABA receptor (This is crucial concerning Tren-Insomnia as healthly function levels of GABA will produce a stable sleep state/environment for rest) and possess, in no particular order, antidepressant,stress-reducing, feeling warm/fuzzy/rewarding,pro-social, anti-aggressive(huge consider tren sides),pro-sexual,sedative/pro-sleep,cognitive-memory improvement..The list goes on!

(Where does this apply with Tren? It can aid all the way around with individuals how are sensitive or not.From the social aspect,overwhelming sense of anxiety,lack of sleep,basically everything stated above that may apply with the usage of tren and the onset of its unwanted side)

In addition to this information, an individual can also utilized masteron (Drostanolonein) in conjunction with Proviron, running both concurrent may yield a great synergenic effect,each compound will compliment one an other.

Below is a image illustrating the neurotransmitter/receptor and how it functions, also I will include some real actual studies conducted with proven results expressing the benefits of this compound (proviron)
Keep in mind that these doses may seem extreme,its been proven time and time again that such significant dosages are not needed to yield the effect. Merely a daily intake of 50-100 will suffice for almost anyone!
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Further more Proviron is a DHT derivative. DHT compounds assist with hardening of the physique, lack of water retention,increased sex drive..Hardening of the physique and lack of water retention go hand in hand. Proviron assists with this, The body recognizes proviron as a DHT,This causes a direct hardening affect on the muscle tissue (Like mast posses,but mast is much more stronger IMO) The increase in hardness comes from a reduction in free estrogen levels, because proviron has the ability to 'latch-on' to the estrogen binding enzymes,It competes so to speak for its position,it does this aggressively, thus decreasing water retention. Also the the lack of aromatization and the fact that the drug is prototypical androgen, causes a significant shift in the body***8217;s estrogen/testosteroneratio.As proviron's atomic structure it is incapable of forming estrogen. It also has properties with AR's.. Increasing the AR expression, proviron/DHT uptake to further increase AR expression, repeating this process over and over ...
This allows other AAS compounds to appear to be amplified with there effects,assisting the compounds - (What does this mean?) It can be a master key so to speak, having multiple functions - It binds aggressively to the AR's and SHBG, thus it can/may increase the activity of other AAS/Androgens in the system) - This is an added bonus!

Functions concerning the neurotransmitter/receptor and how it works:Below is a image illustrating the neurotransmitter/receptor and how it functions, also I will include some real actual studies conducted with proven results expressing the benefits of this compound (proviron)
Keep in mind that these doses may seem extreme,its been proven time and time again that such significant dosages are not needed to yield the effect. Merely a daily intake of 50-100 will suffice for almost anyone!

Citation
Database: PsycINFO
[ Journal Article ]
A comparison of the antidepressant effects of a synthetic androgen (mesterolone) and amitriptyline in depressed men.
Vogel, William; Klaiber, Edward L.; Broverman, Donald M.
Journal of Clinical Psychiatry, Vol 46(1), Jan 1985, 6-8.

Abstract

26 depressed male outpatients were randomly assigned to 14 wks of treatment with either mesterolone or amitriptyline in a double-blind parallel treatment design. Ss completed the Hamilton Rating Scale for Depression and a symptom checklist each week. Findings reveal that the drugs were equally effective in reducing depressive symptoms. Mesterolone produced significantly fewer adverse side effects than amitriptyline and did not produce hypomania or tachycardia, recognized side effects of amitriptyline. (10 ref) (PsycINFO Database Record (c) 2013 APA, all rights reserved)

Methods Find Exp Clin Pharmacol. 1984 Jun;6(6):331-7.
The effects of mesterolone, a male sex hormone in depressed patients (a double blind controlled study).
Itil TM, Michael ST, Shapiro DM, Itil KZ.
Abstract
Based on computer EEG (CEEG) profiles, in high doses, antidepressant properties of mesterolone, a synthetic androgen, were predicted. In a double-blind placebo controlled study, the clinical effects of 300-450 mg daily mesterolone were investigated in 52 relatively young (age range 26-53 years, mean 42.7 years) male depressed outpatients. During 6 weeks of mesterolone treatment, there was a significant improvement of depressive symptomatology. However, since an improvement was also established during the placebo treatment, no statistically appreciable difference in the therapeutic effects of mesterolone was established compared to placebo. Mesterolone treatment significantly decreased both plasma testosterone and protein bound testosterone levels. Patients with high testosterone levels prior to treatment seem to have had more benefit from mesterolone treatment than patients with low testosterone levels. The degree of improvement weakly correlated to the decrease of testosterone levels during mesterolone treatment.

Information confirming no HTPA shutdown/suppression during PCT

These are some research articles that may justify the use of low/moderate dose Proviron during PCT:

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AAKVAAG, A., and S. B. STROMME. "The effect of mesterolone administration to normal men on the pituitary-testicular function."Acta endocrinologica 77.2 (1974): 380-386.

ABSTRACT
Mesterolone (1***945;-methyl-5***945;-dihydrotestosterone) has been given to 10 normal men, age 24***8211;27 years, and the effect on the plasma levels of ICSH, FSH and testosterone has been studied.No effect on the plasma levels of ICSH and FSH could be detected. After 4 weeks on 75 mg mesterolone per day a significant (P < 0.01) drop in the mean value for plasma testosterone level was observed, 5.2 to 4.0 ng/ml. After another 4 weeks on 150 mg mesterolone per day a further decrease to 3.5 ng/ml was found.During mesterolone administration the protein binding of testosterone in plasma was significantly reduced, and it appeared that the level of free (non-protein bound) testosterone in diluted plasma remained unchanged, 0.37 and 0.41 ng/ml, before and after mesterolone administration respectively.The results suggest that mesterolone given in doses of 75 and 150 mg/day to normal men does not suppress the pituitary ICSH production or the testicular testosterone production
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GORDON, R.D., THOMAS, M.J., POYNTING, J.M. and STOCKS, A.E. (1975), Effect of Mesterolone on Plasma L.H., F.S.H. and Testosterone. Andrologia, 7: 287***8211;296. doi: 10.1111/j.1439-0272.1975.tb00942.x

Summary
It has been claimed that orally administered mesterolone, unlike l7a-methyl testo- sterone, does not suppress endogenous gonadotrophins and testesterone. To investi- gate this, both drugs were administered, in turn, to four normal men and plasma te- stosterone, L.H. and F.S.H. were measured serially. Mesterolone administration was associated in all four subjects with significant and similar falls in plasma testosterone, but significant suppression of gonadotrophins took place in only two of them. Any changes which occured were apparent by the end of the first week of therapy. Administration of half the dose of 17a-methyl testosterone to the same four subjects caused significant suppression of testosterone in each and suppression of one or both gonadotrophins in each.
In longer term studies in patients (5-30 months) involving serial measurements at intervals of one to two months, there was evidence of significant suppression of L.H. and F.S.H. by 17a-methyl testosterone, but not by mesterolone, which was clinically a less effective androgen.
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WANG, C., BURGER, H.G., de KRETSER, D.M., DULMANIS, A., HUDSON, B., KEOGH, E.J. and SUTHERS, M.B. (1974), Effect of Mesterolone on Serum FSH, LH and Plasma Testosterone in Normal Men. Andrologia, 6: 111***8211;117. doi: 10.1111/j.1439-0272.1974.tb01604.x

Summary
To determine whether the claim that mesterolone, an orally active androgen, does not cause suppression of gonadotrophin secretion, two groups of five normal men were treated with 100 and 200 mg. daily respectively for 7 days. Serial measurements of serum FSH, LH and plasma testosterone were made on samples taken at 15 minute intervals over 2 hr both before and during treatment. Modest falls in FSH, LH and testosterone levels were observed in both groups, the percentage suppression being 21% and 18% for FSH, 19% and 15% for LH and 9% and 8% for testosterone at the lower and higher dosage levels respectively.
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200mg is a far greater dose than I would deploy during PCT (50mgs is ideal). From these studies and other articles we see have read, it's clear that there is almost minimal/no influence on the HTPA, any effect would be absolute minimal and negated by other appropriate compounds used during that period (HCG, Aromasin, Nolvadex and HGH)...

Vision

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I'd also like to add some stellar information about "provirion" and its amazing proprieties..

Provirion won't suppress LH or FSH and will even provide your body with more free testosterone due to the fact that it binds "AGGRESSIVELY" to SHBG (lowering levels)....With this said it's not suppressive like most steroids, and your FREE T can greatly increase by allowing bound test to act in its original course of action..in lieu of being bound (freeing up bound test)

Proviron will assist in a blast/cycle
It's already known that only a very small amount of Testosterone exists as free testosterone. Free test is testosterone that capable of binding to the Androgen Receptor, which is where all the rest of the magic happens, and allows for the following benefits:
-Enhanced growth factor activity (e.g. GH, IGF-1, etc.)
-Enhanced activation of myogenic stem cells (i.e. satellite cells)
-Enhanced myonuclear number (to maintain nuclear to cytoplasmic ratio)
-Enhanced protein synthesis
-New myofiber formation

Testosterone binds at around 45% to what is known as Sex Hormone Binding Globulin (SHBG), and about another 53% binds to proteins (albumin). The rest exists in a free state (about 2% if you did your math). Different variations of steroids also differ in the way in which they bind to proteins.
If one could unbind testosterone from SHBG by even a small percentage, it could make a big difference in the way that testosterone or other AAS exert their anabolic effects. Studies show that when testosterone is unbound from SHBG the free test does in fact exert greater effects than total T. As the following studies support:

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Demisch K, and Nickelsen T. Distribution of testosterone in plasma proteins during replacement therapy with testosterone enanthatein patients suffering from hypogonadism Andrologia 1983;15 Spec No:536-41.

Gandar R. Interpretation of the blood level of a steroid Rev Fr Gynecol Obstet 1985 Aug-Sep;80(8-9):635-40.

Legrand E., et al. Osteoporosis in men: a potential role for the sex hormone binding globulin Bone 2001 Jul;29(1):90-5.

Longcope C., et al. Diet and sex hormone-binding globulin. J Clin Endocrinol Metab 2000 Jan;85(1):293-296.

Valero-Politi J, and Fuentes-Arderiu X. Within- and between-subject biological variations of follitropin, lutropin, testosterone, and sex-hormone-binding globulin in men. Clin Chem 1993 Aug;39(8):1723-1725.

Proviron(1-Methyl Dihydrotestosterone) has been shown to bind with SHBG much more readily than test.

Relative binding affinity of anabolic-androgenic steroids: comparison of the binding to the androgen receptors in skeletal muscle and in prostate, as well as to sex hormone-binding globulin.

Saartok T, Dahlberg E, Gustafsson JA.

It is unclear whether anabolic steroids act on skeletal muscle via the androgen receptor (AR) in this tissue, or whether there is a separate anabolic receptor. When several anabolic steroids were tested as competitors for the binding of [3H]methyltrienolone (MT; 17 beta-hydroxy-17 alpha-methyl-4,9,11-estratrien-3-one) to the AR in rat and rabbit skeletal muscle and rat prostate, respectively, MT itself was the most efficient competitor. 1 alpha-Methyl-5 alpha-dihydrotestosterone (1 alpha-methyl-DHT; mesterolone) bound most avidly to sex hormone-binding globulin (SHBG) [relative binding affinity (RBA) about 4 times that of DHT]. Some anabolic-androgenic steroids bound strongly to the AR in skeletal muscle and prostate [ RBAs relative to that of MT: MT greater than 19-nortestosterone ( NorT ; nandrolone ) greater than methenolone (17 beta-hydroxy-1-methyl-5 alpha-androst-1-en-3-one) greater than testosterone (T) greater than 1 alpha-methyl-DHT]. In other cases, AR binding was weak (RBA values less than 0.05):stanozolol(17 alpha-methyl-5 alpha- androstano [3,2-c]pyrazol-17 beta-ol), methanedienone (17 beta-hydroxy-17 alpha-methyl-1,4-androstadien-3-one), and fluoxymesterolone (9 alpha-fluoro-11 beta-hydroxy-17 alpha-methyl-T). Other compounds had RBAs too low to be determined (e.g. oxymetholone (17 beta-hydroxy-2-hydroxymethylene-17 alpha-methyl-5 alpha-androstan-3-one) and ethylestrenol (17 alpha-ethyl-4- estren -17 beta-ol). The competition pattern was similar in muscle and prostate, except for a higher RBA of DHT in the prostate. The low RBA of DHT in muscle was probably due to the previously reported rapid reduction of its 3-keto function to metabolites, which did not bind to the AR [5 alpha-androstane-3 alpha, 17 beta-diol and its 3 beta-isomer (3 alpha- and 3 beta-adiol, respectively)]. Some anabolic-androgenic steroids (only a few synthetic) bound to SHBG (1 alpha-methyl-DHT much greater than DHT greater than T greater than 3 beta-adiol greater than 3 alpha-adiol = 17 alpha-methyl-T greater than methenolone greater than methanedienone greater than stanozolol). The ratio of the RBA in rat muscle to that in the prostate (an estimate of the myotrophic potency of the compounds) was close to unity, varying only between about 0.4 and 1.7 in most cases.(ABSTRACT TRUNCATED AT 400 WORDS)

Skalba P, Korfanty A, Mroczka W, Wojtowicz M. Related Articles
[Changes of SHBG concentrations in postmenopausal women]
Ginekol Pol. 2001 Dec;72(12A):1388-92. Polish.

Variations of sex hormone-binding globulin in thyroid dysfunction.

Brenta G, Schnitman M, Gurfinkiel M, Damilano S, Pierini A, Sinay I, Pisarev MA.

Department of Endocrinology and Metabolism, French Hospital, Buenos Aires,
Argentina.brenta@cnea.gov.ar


With the aim of understanding the variations of the levels of sex hormone-binding globulin (SHBG) in thyroid dysfunction, we studied the influence of factors that also modify SHBG, such as menopausal status, age, and body mass index (BMI) in women with hypothyroidism and hyperthyroidism, both overt and subclinical. Statistical analysis was performed by means of analysis of variance (ANOVA), stepwise multiple regression, and partial correlation. The ANOVA showed a significant statistical difference among the means of SHBG of all groups (p<0.01). The difference was due to the group that included hyperthyroid women. Multiple regression analysis showed that the main factors influencing SHBG were BMI and age, except for the hyperthyroid group, where the most important independent variables were triiodothyronine (T3) and thyroxine (T4). Partial correlation controlling the effect of BMI and age showed no association between SHBG and the other variables in all groups except for the subclinical hyperthyroid and hyperthyroid, where we found a significant association between SHBG and T4 and T3. The premenopausal or postmenopausal status did not modify SHBG levels. When the patients are taken as a whole, BMI, age, T4, and T3 all have an association with SHBG levels according to the multiple regression analysis.

Determinants of sex hormone-binding globulin blood concentrations in premenopausal and postmenopausal women with different estrogen status. Virgilio-Menopause-Health Group.

Pasquali R, Vicennati V, Bertazzo D, Casimirri F, Pascal G, Tortelli O, Labate AM.

Department of Internal Medicine and Gastroenterology, University of Bologna, Italy

Just a quote: 2) SHBG values are correlated positively with estradiol and negatively withinsulin and testosterone concentrations, but the predictive value of these variabiles on SHBG appears to be different in premenopause and postmenopause;

Here is a fun little fact: the level of SHBG can also be influenced by other factors. There is a direct relationship between the level of estrogen and thyroid hormones and the level of SHBG. Estrogen goes up, SHBG goes up. Estrogen goes down SHBG goes down. Same for Thyroid hormones triiodothyronine (T3) and thyroxine (T4). Also, there is a relationship with diet and insulin, but that is something I will save for later. Higher androgen levels due to AS administration has been shown to considerably lower levels of SHBG as well. The ASWinstrol (stanozolol) was shown in a 1989 study to lower levels of SHBG by 50% after oral administration.


Sex hormone-binding globulin response to the anabolic steroid stanozolol: evidence for its suitability as a biological androgen sensitivity test.

Sinnecker G, Kohler S.

Department of Pediatrics, University of Hamburg, West Germany.


Both the androgen-induced decline in serum sex hormone-binding globulin (SHBG) levels during puberty and the anabolic effect of exogenous testosterone are absent in patients with androgen insensitivity (testicular feminization). To determine whether the androgen-induced decline in serum SHBG could be used as a test of androgen sensitivity, we studied the effect of the anabolic-androgenic steroid stanozolol (17 beta-hydroxy-17 alpha-methyl-5 alpha-androstano-[3,2-c]pyrazol) on serum SHBG in 25 control subjects, 3 patients with complete androgen insensitivity, and 4 patients with partial androgen insensitivity. Stanozolol was administered orally for 3 days (0.2 mg/kg.day); blood samples were taken before and 5, 6, 7, and 8 days after the beginning of the test for measurements of serum SHBG. The lowest value (i.e. the peak response) in each subject was used as the measure of the response to stanozolol. In the control subjects the mean nadir serum SHBG level was 51.6 +/- 5.9% (+/- SD) of the initial value (P less than 0.001). In the 4 patients with partial androgen insensitivity the nadir serum SHBG ranged from 73-89%, and in the 3 patients with complete androgen insensitivity it ranged from 93-97% of the initial value. Thus, the decrease in serum SHBG after short term administration of stanozolol reflects androgen responsiveness and, thus, may be used to differentiate patients with androgen insensitivity syndromes from those with other causes of male pseudohermaphroditism.

Proviron could provide the mechanisms to increase the value of other AS... Proviron would work because by binding to SHBG, it leaves hormone in a free state to bind to the AR. Proviron is a terrible Anabolic, but its affinity for SHBG would essentially displace other steroids from binding to SHBG...Provirion would work to reduce the overall amount of SHBG, thereby having the effect of freeing up hormone to bind to the AR.

(Click here to order your Proviron TODAY)

This info is actually pretty good timing because I'm using presently using Masteron for the first time, but Proviron is another compound that i haven't used in the past, and I plan to include it in my next cycle
 
This info is actually pretty good timing because I'm using presently using Masteron for the first time, but Proviron is another compound that i haven't used in the past, and I plan to include it in my next cycle

It's a lot of information to read but it's up there so you can come back and chip away at it in the future..you would be presently surprised on how well these work with Trenbolone..it's not a miracle but it gives you the added advantage..
 
It's a lot of information to read but it's up there so you can come back and chip away at it in the future..you would be presently surprised on how well these work with Trenbolone..it's not a miracle but it gives you the added advantage..
For me it just kind of levels me off when on tren. Masteron is a happy drug for me....tren is not.
 
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