I know it doesn't work this way but not sure why. Wouldn't that be nice if it did.
So if serms work by blocking the estrogen receptors in the pituitary, thereby tricking it into thinking the body needs more estrogen, therefore triggering an LH response so the body makes more testosterone to be converted to estrogen,.. How come when estrogen levels are normal, even during a high testoerone cycle, does the pituitary not produce a normal amount of LH?
So if serms work by blocking the estrogen receptors in the pituitary, thereby tricking it into thinking the body needs more estrogen, therefore triggering an LH response so the body makes more testosterone to be converted to estrogen,.. How come when estrogen levels are normal, even during a high testoerone cycle, does the pituitary not produce a normal amount of LH?