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Got Fat? Pass the Testosterone Please! (Study Included)

Vision

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Get Shredded!
There's been some discussions lately in regards to Testosterone and fat loss..Or Testosterone only cycles vs people advocating BF% must be low before starting a FIRST cycle, or fat loss cycle ext..
I've stressed from time and time again about the bro-science behind "Lower your body fat before your first cycle"..
Below is some illustrations compiled of reads/studies that I have gathered behind the benefits of testosterone and fat loss..



Team Supervisor SF
Vision

______________________________________-

The first illustration is a study conducted with TRT

Testosterone therapy in hypogonadal men results in sustained and clinically meaningful weight loss.


Yassin A1, Doros G.
Author information



Abstract

WHAT IS ALREADY KNOWN ABOUT THIS SUBJECT:

Hypogonadism is associated with increased fat mass and reduced muscle mass, which contributes to obesity and health risks, such as cardiovascular disease.Testosterone treatment of hypogonadal men improves muscle mass and reduces fat mass; however, many of these studies are of short duration.Thus, the long-term effects of testosterone on body anthropometry are not known.
WHAT THIS STUDY ADDS:

Long-term testosterone treatment of hypogonadal men, up to 5 years duration, produced marked and significant decrease in body weight, waist circumference and body mass index. Hypogonadism contributes to reduced muscle mass and increased adiposity.Testosterone treatment ameliorates loss of muscle mass and reduces fat accumulation associated with hypogonadism. In this study, we evaluated the long-term effects of normalizing testosterone (T) levels in hypogonadal men on anthropometric parameters. Open-label, single-center, cumulative, prospective registry study of 261 men (32-84 years, mean 59.5 ± 8.4 years, with T levels ≤12 nmol L-1 [mean: 7.7 ± 2.1]). Among the 261 men on T treatment, we followed up on 260 men for at least 2 years, 237 for 3 years, 195 for 4 years and 163 for at least 5 years. Subjects received parenteral T undecanoate 1000 mg every 12 weeks after an initial interval of 6 weeks. Body weight (BW), waist circumference (WC) and body mass index (BMI) were measured at baseline and yearly after treatment with T. BW decreased from 100.1 ± 14.0 kg to 92.5 ± 11.2 kg and WC was reduced from 107.7 ± 10.0 cm to 99.0 ± 9.1 cm. BMI declined from 31.7 ± 4.4 m kg-2 to 29.4 ± 3.4 m kg-2. All parameters examined were statistically significant vs. baseline and vs. the previous year over 5 years, indicating a continuous weight loss (WL) over the full observation period. The mean per cent WL was 3.2 ± 0.3% after 1 year, 5.6 ± 0.3%, after 2 years, 7.5 ± 0.3% after 3 years, 9.1 ± 0.3% after 4 years and 10.5 ± 0.4% after 5 years. The data obtained from this uncontrolled, observational, registry study suggest that raising serum T to normal physiological levels in hypogonadal men produces consistent loss in BW, WC and BMI. These marked improvements were progressive over the 5 years of the study.


KEYWORDS:

Testosterone, obesity, waist circumference, weight loss

PMID: 24163704 [PubMed] PMCID: PMC3799011 Free PMC Article

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Got Fat? Pass the Testosterone Please!


Consider this: how many times have you read or heard that there is no such thing as a “cutting” steroid? For years the generally accepted doctrine was that steroids do not reduce adipose tissue but rather can decrease water retention and inhibit estrogen’s actions thus producing the “hard” look. Furthermore, the consensus seems to be that while testosterone and related anabolic substances certainly aid the dieting bodybuilder it is only via the androgen’s anti-proteolytic abilities, thus sparing muscle tissue while in a hypocaloric state, in addition to the aforementioned “drying out” effect.

In short – this is hogwash!! As this article will validate, an abundance of clinical research and peer-reviewed data strongly supports testosterone’s (T) fat reducing actions and its preventative impact on adipocyte generation (1,2,3). Therefore, T acts both in the breakdown of existing fat tissue and to hinder pre-adipocytes from maturing. CCAAT enhancer-binding proteins (CEBPs) are vital contributors at several stages of adipogenesis – or fat cell formation – from early uptake and accumulation of lipids to the differentiation, proliferation, and terminal production of the adipocyte. CEBPs exist in an alpha, sigma, and beta form. The beta and sigma form promote the transcription of PPARy2, which is arguably the most important factor in fat cell formation. CEBP possesses intrinsic pro-adipogenic activity independent of PPARy2. Specifically, the alpha and sigma isoform of CEBP interact with promoter regions of DNA involved in adipose development (5). PPARy2, stimulated by the CEBP isoforms, is capable of the same. This leads to a viscous cycle beginning with CEBP activity. DNA promotion of said adipogenic proteins follows, including PPARy2, which itself then activates the same lipid-related genes as the CCAAT enhancer-binding proteins. Testosterone has been shown to reduce all three forms of CEBP’s, which, predictably, curtails PPARy2 functions (5, 6, 7, 17, 23). Evidence exists as well that dihydrotestosterone shares T’s anti-CEBP characteristics (6, 23). Like muscle tissue, fat cells express anabolic receptors, which bind to respective androgens and initiate inhibitory effects on CEBP’s. Visceral Adipose Tissue (VAT) is more sensitive to T than subcutaneous fat (SC). This is most likely explained by the fact that AR’s occur in greater number in VAT versus SC tissue (2, 3, 4). T’s strong effect on VAT coincides with the observed reduction in abdominal fat in hypogonadal men placed on testosterone therapy. Furthermore, T’s antiadiogenic and lipolytic actions are more predominant in VAT (1, 2). Pre-adipocytes, located in VAT, show reduced activity of the CEBP’s when exposed to testosterone (5, 6). A novel observational study followed 511 males aged 30-79, over a 12-year period charting the effects of serum androstenedione, testosterone, and SHBG and their relation to VAT. Hormone analysis was conducted before and after this period and concluded that below normal androstenedione and testosterone show strong correlation with abdominal adipose tissue. (8). Peroxisomal proliferator-activated receptor gamma 2 is another protein that functions to increase the differentiation of adipocytes. Treatment with either testosterone or DHT reduces PPARg2 providing another means whereby androgens discourage the differentiation stage in adipogenesis (22) The metabolic alterations experienced in VAT due to testosterone are of significance with both negative and positive ramifications. Lipid uptake is inhibited in VAT mainly due to a decrease in lipoprotein-lipase activity, and a reduction in serum insulin and glucose levels (9). It should be noted that lipid uptake is enhanced by elevated hyperglycemia/hyperinsulinemia, primarily in VAT (10). So the decrease in VAT noted with a reduction of glucose/insulin levels – via testosterone elevation – should make sense. The ability of testosterone to stimulate fatty acid release from VAT can be problematic, though. Feeding directly into the liver via the portal vein, these fatty acids have the potential to impair both insulin sensitivity and function, induce hyperglycemia, and increase LDL levels. Ultimately this can lead to hyperinsulinemia, hyperglycemia, hyperlipidemia, and hypertension, vascular resistance, all of which are consistent with the pathology of “Metabolic Syndrome X” (11). In addition, cortisol, originating in VAT, is also released and fed to the liver via the portal vein. This exacerbates fat cell maturity as cortisol increases the differentiation of pre-adipocytes into the adult forms (13). It would seem logical then to suggest a cause and effect relationship between free fatty acid release from VAT by testosterone and the development the conditions of Metabolic Syndrome X. This may be a moot point though as testosterone treatment has been associated with an actual reduction in both total cholesterol and LDL levels. Previous studies indicate that testosterone – especially its chemically altered versions – can lower HDL levels deemed the “good cholesterol”. This has not been demonstrated conclusively, as results of other research efforts imply that T is neutral in regards to HDL, neither causing a significant increase or decrease (12, 16). In relation to the cardiovascular disorders, normal or slightly enhanced T levels might exert a protective effect. Pro-inflammatory cytokines TNF , IL-1ß, and IL-6 in human macrophages, are positively associated with congestive heart failure and atherosclerosis. Not only has testosterone been shown to reduce the elevations in these cytokines but also up regulates anti-inflammatory cytokines such as IL-10, leading to an improvement in improving overall cardiovascular function. (12, 14, 15) This decreases susceptibility to congestive heart failure and vascular disease also due in part to T’s vasodilating characteristics.In relation to existing fat tissue, T levels are inversely associated with lipoprotein lipase, an enzyme involved in the breakdown of triglycerides and recycling of the resulting fatty acids and eventual production a mature lipid-rich adipocyte. Testosterone also impairs overall lipid uptake into fat cells, hindering hypertrophy of existing fat cells. T is positively correlated with beta receptor number catecholamine sensitivity, thus heightening thermogenesis (18, 19, 20). The thermogenic process involves a G-protein activation and a downstream production of lipolytic enzymes and proteins. Both DHT and Testosterone amplifies adenylate cyclase activity, an enzyme vital to beta receptor stimulated lipolysis (21). Moreover, hormone-sensitive lipase (HSL) initiates the breakdown of stored tryiglycerides into fatty acids, which can be oxidized and thus “burned” in either mitotochondria and/or peroxisomes. T not only elevates HSL but also Protein Kinase A, an enzyme that functions in the G-protein stimulated lipolysis (18, 20) By jacking up these lipolytic enzymes and proteins, beta oxidation occurs. Androgens also increase other such enzymes including carboxylesterase 3, acetyl-coenzyme A acyltransferase 1, 3-ketoacyl-CoA thiolase B and enoyl-coenzyme A hydratase/3-hydroxyacyl coenzyme A dehydrogenase (2, 24). Enzymes vital to lipogenesis, conversely, show a consistent negative correlation with both T and DHT. Malic enzyme, glyceraldehyde dehydrogenase (GDH), and fatty acid elongation factors – all capable of pro-lipogenic effects, are inhibited by DHT and T (2, 24) It is of great importance, though, to understand that the T’s positive benefits espoused in this article DO NOT apply to females. In fact, typically, the majority of natural male androgens, such as testosterone and DHT, exert converse or opposing actions – often negative – in females compared to males. Furthermore, the studies and research cited herein refer to inherent activity of endogenous T and DHT or hormone replacement therapy (HRT). These intrinsic natural androgens and their actions, thus, do not extend to chemical altered androgens or anabolic steroids. This does not necessarily imply that AAS do not have the potential to mimic endogenous male hormone. This just indicates that only research reviews examining naturally produced androgens were considered herein. If truth be told, a follow-up article delving even deeper into functions of T on obesity would certainly be justified. It also could review effects of both testosterone in females and chemically altered androgens. This article provides a fairly brief review of what is actually an impressive and growing body of evidence supporting Testosterone and other androgens as potent lipolytic and anti-adipogenic hormones. In fact, while the author had reviewed relevant literature beforehand describing the interactions of androgens and adipose, the additional dissection of this topic revealed a much greater than anticipated number of mechanisms and actions of natural androgens. Testosterone has not really received the respect and appreciation it deserves for its wide range of health benefits. Recently, though, with the ever-increasing administration of HRT to males and evidence confirming its potential benefits, the negative connotation and exaggerated drawbacks of such treatment is dying out. In its place exists a wealth of legitimate research claims and an increased priority placed on unbiased future studies.References

1.Peter J. Snyder, Helen Peachey, Peter Hannoush, Jesse A. Berlin, Louise Loh, David A. Lenrow, John H. Holmes, Abdallah Dlewati, Jill Santanna, Clifford J. Rosen and Brian L. Strom. Effect of Testosterone Treatment on Body Composition and Muscle Strength in Men Over 65 Years of Age1The Journal of Clinical Endocrinology & Metabolism Vol. 84, No. 8 2647- \
2.Hutley L, Joyner J Cameron D. Intrinsic Horm Metab Res. 2002 May;34(5):223- regional differences in androgen receptors and dihydrotestosterone metabolism in human preadipocyte. 3. M. N. Dieudonné, R. Pecquery, A. Boumediene, M. C. Leneveu, and Y. Giudicelli. Androgen receptors in human preadipocytes and adipocytes: regional specificities and regulation by sex steroids Am J Physiol Cell Physiol 274: C1645-C1652, 1998; 4 Androgen hormone binding to adipose tissue in rats. Sjogren J, • Li M, • Bjorntorp P. Biochim Biophys Acta. 1995 May 11;1244(1):117–120 5 Rajan Singh, Jorge N. Artaza, Wayne E. Taylor, Melissa Braga, Xin Yuan, Nestor F. Gonzalez-Cadavid and Shalender Bhasin Testosterone Inhibits Adipogenic Differentiation in 3T3-L1 Cells: Nuclear Translocation of Androgen Receptor Complex with ß-Catenin and T-Cell Factor 4 May Bypass Canonical Wnt Signaling to Down-Regulate Adipogenic Transcription Factors. Endocrinology Vol. 147, No. 1 141-154http://endo.endojournals.org/cgi/content/abstract/147/1/141?ck=nck 6. Rajan Singh, Jorge N. Artaza, Wayne E. Taylor, Nestor F. Gonzalez-Cadavid and Shalender Bhasin. Androgens Stimulate Myogenic Differentiation and Inhibit Adipogenesis in C3H 10T1/2 Pluripotent Cells through an Androgen Receptor-Mediated Pathway. Endocrinology, doi:10.1210/en.2003-0741 7. Shalender Bhasin, Thomas W. Storer, Nancy Berman, Kevin E. Yarasheski, Brenda Clevenger, Jeffrey Phillips, W. Paul Lee, Thomas J. Bunnell and Richard Casaburi. Testosterone Replacement Increases Fat-Free Mass and Muscle Size in Hypogonadal Men1. The Journal of Clinical Endocrinology & Metabolism Vol. 82, No. 2 407-413 8. Khaw KT, Barrett-Connor E. Lower endogenous androgens predict central adiposity in men. Horm Metab Res. 2002 May;34(5):223-8 9. P Marin, B Oden and P Bjorntorp Assimilation and mobilization of triglycerides in subcutaneous abdominal and femoral adipose tissue in vivo in men: effects of androgens. Journal of Clinical Endocrinology & Metabolism, Vol 80, 239-243, 10. Marin P, Andersson B, Ottosson M, Olbe L, Chowdhury B, Kvist H, Holm G, Sjostrom L, Bjorntorp P. The morphology and metabolism of intraabdominal adipose tissue in men. Metabolism. 1992 Nov;41(11):1242-8. 11. Bergman RN, Kim SP, Hsu IR, Catalano KJ, Chiu JD, Kabir M, Richey JM, Ader M. Abdominal obesity: role in the pathophysiology of metabolic disease and cardiovascular risk. Am J Med. 2007 Feb;120(2 Suppl 1):S3-8; discussion S29-32. 12. Chris J. Malkin, Peter J. Pugh, Richard D. Jones, Dheeraj Kapoor, Kevin S. Channer and T. Hugh Jones. The Effect of Testosterone Replacement on Endogenous Inflammatory Cytokines and Lipid Profiles in Hypogonadal Men. The Journal of Clinical Endocrinology & Metabolism Vol. 89, No. 7 3313-3318 13. Ruth Andrew1, Jukka Westerbacka2, John Wahren3, Hannele Yki-Järvinen2, and Brian R. Walker1. The Contribution of Visceral Adipose Tissue to Splanchnic Cortisol Production in Healthy Humans. Diabetes 54:1364-1370, 2005 14. Cutolo M, Balleari E, Giusti M, Intra E, Accardo S. Androgen replacement therapy in male patients with rheumatoid arthritis. Arthritis Rheum. 1991 Jan;34(1):1-5. 15. P J Pugh1, R D Jones2, J N West1, T H Jones2 and K S Channer1. Testosterone treatment for men with chronic heart failure. Heart 2004;90:446-447 16. D Kapoor1,3, E Goodwin1, K S Channer2 and T H Jones1,3. Testosterone replacement therapy improves insulin resistance, glycemic control, visceral adiposity and hypercholesterolaemia in hypogonadal men with type 2 diabetes. European Journal of Endocrinology, Vol 154, Issue 6, 899-906 17. E Garcia, M Lacasa, B Agli, Y Giudicelli, and D Lacasa. Modulation of rat preadipocyte adipose conversion by androgenic status: involvement of C/EBPs transcription factors. Journal of Endocrinology, Vol 161, Issue 1, 89-97 18. XF Xu, G De Pergola and P Bjorntorp. Testosterone increases lipolysis and the number of beta-adrenoceptors in male rat adipocytes. Endocrinology, Vol 128, 379-382, 19. Rebuffé-Scrive M, Mårin P, Björntorp P. Effect of testosterone on abdominal adipose tissue in men. Int J Obes. 1991 Nov;15(11):791-5. 20. X Xu, G De Pergola, PS Eriksson, L Fu, B Carlsson, S Yang, S Eden and P Bjorntorp. Postreceptor events involved in the up-regulation of beta-adrenergic receptor mediated lipolysis by testosterone in rat white adipocytes. Endocrinology, Vol 132, 1651-1657 21. Pecquery R, Dieudonne MN, Leneveu MC, Giudicelli Y. Evidence that testosterone modulates in vivo the adenylate cyclase activity in fat cells. Endocrinology. 1990 Jan;126(1):241-5 22. Singh R, Artaza JN, Taylor WE, Gonzalez-Cadavid NF, Bhasin S. Androgens stimulate myogenic differentiation and inhibit adipogenesis in C3H 10T1/2 pluripotent cells through an androgen receptor-mediated pathway. Endocrinology. 2003 Nov;144(11):5081-8. Epub 2003 Jul 24. 23. M. N. Dieudonne, R. Pecquery, M. C. Leneveu and Y. Giudicelli. Opposite Effects of Androgens and Estrogens on Adipogenesis in Rat Preadipocytes: Evidence for Sex and Site-Related Specificities and Possible Involvement of Insulin-Like Growth Factor 1 Receptor and Peroxisome Proliferator-Activated Receptor 2. Endocrinology Vol. 141, No. 2 649-656 24. Dieudonne MN, Pecquery R, Leneveu MC, Giudicelli Y. Opposite effects of androgens and estrogens on adipogenesis in rat preadipocytes: evidence for sex and site-related specificities and possible involvement of insulin-like growth factor 1 receptor and peroxisome proliferator-activated receptor gamma2. Endocrinology.

 
The black is a better athlete to begin with because he’s been bred to be that way — because of his high thighs and big thighs that goes up into his back, and they can jump higher and run faster because of their bigger thighs. This goes back all the way to the Civil War when during the slave trading, the owner — the slave owner would breed his big black to his big woman so that he could have a big black kid.

10.jpg
 
Get that nonsense out of my thread!
 
Never understood how the whole "africans have the best genes" racism thing. I don't see how complimenting an entire race at being physically superior to us skinny small cocked white boys is being a racist with negative connotations. I guess when you get into the reasons why they are you are on a thin line of a lot of assumption. Either way, when it comes to sports, muscle building, speed African and high echelon German genetics are right there at the top.
 
Lets not forget the Swedes or Russians either..
 
Maybe because races comprise sports they have interest in. I don't think you can say Africans have better genes for basketball compared to skiing, it's just that's the sport they are most interested in. If more white guys played basketball instead of Hockey, I think it would even out.
 
back on topic, this was a good article. I will think about it every time I see the late night commercials for lawsuits against TRT docs.
 
My TRT dose of 150mg has me losing mid section fat and gaining mass. That alone tells me these are accurate studies and everytime I took a lot of test, I got leaner without a perfect diet mind you.
 
Most of this is basics, but other often look/resort to other compounds before actually knowing what possibilities Testosterone posses...

If any newbies are reading this and have already made up your mind about using AAS, and your goal is to lose weight?! Stay away from the profiles in regards to other agents/compounds and focus on diet and Test only..Basics can yield exceptional results, basics are not only ideal, but basics always win...Ask any coach in any sport!
 
Several Testosterone studies show a reduction in body fat. Even more interesting they show body fat is lowered in a dose dependent relationship of Testosterone administration.
 
Yeah - its a shame that the anti-trt suits are putting pressure on the industry because some dingleberry Dr's aren't checking the health of their elderly cardiac patients first.

--Life is a box of chocolates. Lick all the nuts.
 
Absolutely Heavy...Its now proven that TRt and hormones/AAS for that matter yields healthy results,But yet the big mac and whopper are still legal? The irony
 
To this day my best cycle was my first test only cycle. And I feel like a new man since starting trt!
 
TRT and this board changed my life.

Vision, it's Reps like you and the information that you provide, that make this board what it is. Especially lately with all the BS going on, particularly with people from IMF. Don't understand it as it appears we all have a common interest.

At any rate, good info and much appreciated! Always know who to follow when I need to broaden my knowledge.

LowTman
 
Get Shredded!
You can't ban McDonalds. Dlats would be dissapoint.
I will admit I was disappointed when McDonalds discontinued their swiss Angus burger. Because of that I hope every location is sitting above giant sinkhole
 
its members like you with feedback like this that gives me even more reason to contribute with such useful information that everyone could learn from
TRT and this board changed my life.

Vision, it's Reps like you and the information that you provide, that make this board what it is. Especially lately with all the BS going on, particularly with people from IMF. Don't understand it as it appears we all have a common interest.

At any rate, good info and much appreciated! Always know who to follow when I need to broaden my knowledge.

LowTman
 
Absolutely Heavy...Its now proven that TRt and hormones/AAS for that matter yields healthy results,But yet the big mac and whopper are still legal? The irony

Vision,

Appreciate the time and effort to present this article. I would however add a disclaimer to the above statement .... especially for the uninitiated, uneducated or just new to AAS/hormones. "TRT and AAS/Hormomes CAN produce healthy results WHEN used in proper doses by an individual that has considered all factors such as age, over all health, diet and the individual and synergistic effect of each drug they are considering. ANY drug can have beneficial or consequential effects once ALL the facts are considered."

You would think that would go w/o saying .... but its alot of the young guys who will only read "hormones ... AAS ... healthy .... results." .... lol ... I know ..... I used to be one of them.

After 16 years doing this, I wish I had been a little smarter and a little better guided in those first few years. It was only after learning more about pharmacology and biology that I became more wiser.

Just my.02 .... Not trying to high jack the thread or diminish what you are saying as I think you are a great contributor to this board.

I hope that was helpful.

QZ
 
Stellar observation, and good call..Its wishful thinking expecting others to already have an understanding considering the environment we're in..With that being said I've been baffled at times from some of the things I've read on the panels across the forums, so its much of the wiser to be specific with details like you stated above...

Your "example" template-disclaimer above supports and addresses what needs to be clarified for those who lack the understanding and knowhow in regards to AAS..

Thank you for looking out for others brother..
 
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lol .... no way .... medical field man. :winkfinger:

You know anyone who has been around these MB's long enough knows that the most frustrating thing is to see the same questions asked over and over and to see a complete lack of education or at least sensibility about what we do. It's those that are blind to the facts, want a quick fix and uneducated that fail in this game. I do well at what I do ... because i respect it. I approach it like work.

I am glad I joined this board. Lots of good people here. and I mean that. HeavyIron Posts are worth the read. everyone. And I have read alot of them. Its posts like his (and this one by vision) that should be mandatory reading before certain questions are even asked. I love articles. I love scientific reports. I love educated, well thought out questions and the discussion it invokes.

I may sound too serious and even like an asshole at times .... but its because I WANT people to WANT to learn about this shit and what it does and how it does it. It makes the journey so much more worth it when you are educated on the facts. But hey .... that's what happens when you get older .... ha ha ha. i was young once too. I failed many times in beginning because I didn't understand the how's and why's. Once I did ..... sky was the limit and I have reaped the rewards ever since.

QZ
 
Vision,

I am glad you saw the message I was trying to convey and your post says you certainly did. I had no doubt from your past posts I have read that you would understand where I am coming from.

It is only through education and a little trial and error on each individuals part that we gain the knowledge to correctly approach what we do.

If I wanted to fly a plane ..... I wouldn't just jump in the cockpit and try to fly. I would take lessons. Study. Then be ready to take the wheel. Its not much different with AAS (at least in my eyes) because we are playing with our health and our lives. And if you think for a second your aren't .... you shouldn't be doing this. Show it the respect it deserves.

I guess its only because I am in the medical community that perhaps I am a little more sensitive to this subject ... idk.

I apologize for steering the thread off subject from what is a great article. I get a little carried away. :(

/hijack

QZ
 
I know that feeling QZ. Reading is knowledge, knowledge is power, power means success. Its old, trite, and laughed at by today's youth (and yesteryear's as well, let's be honest), but it is true.

Thanks for the posts, QZ and Vision.

--Life is a box of chocolates. Lick all the nuts.
 
bumping this, I've seen a few posts recently with questions and concerns about running his first cycle which multiple compounds and trying to lose body fat. I hope these individuals read this
 
bumping this, I've seen a few posts recently with questions and concerns about running his first cycle which multiple compounds and trying to lose body fat. I hope these individuals read this

Vision this is an excellent article brother. It confirms what test has been doing for me. Before I starting TRT I was really struggling to keep my BF down and get lean. I was by no means a lard ass as I have been lifting for over 20 years, but as I got older keeping BF in check ( despite exercise and better than average diet) was starting to be an uphill battle. Just prior to TRT I was doing a ton of cardio and starvation type bullshit diets. After starting TRT 200 mg PW all that changed. I started eating again and backed off on the cardio some. At first I got nervous because the scale started shooting up. I gained about 20 lbs in 7 months BUT my waist got smaller I got leaner and I put on a lot more muscle. Right now weight wise I am at the high side for me but I have never been leaner in my life. Now do I have that nice 6 pack that I WILL GET SOMEDAY? No but I'm defiantly a step closer and testosterone as a lot to so with it!
 
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